Researchers identify first molecular steps that lead to pancreatic cancer .

By identifying the molecular starting point when certain cells in the pancreas become pre-cancerous lesions, researchers behind a new study believe they have opened the door to exploring ways to prevent the deadly disease.

Study leader Dr. Peter Storz, a cancer biologist at the Mayo Clinic in Jacksonville, FL, who – with colleagues – describes the findings in the journal Cancer Discovery, says:

“Pancreatic cancer develops from these lesions, so if we understand how these lesions come about, we may be able to stop the cancer train altogether.”

Pressing need for new treatments and ways to prevent pancreatic cancer

Location of the pancreas
Cancer of the pancreas is one of the most aggressive human cancers and is the fourth leading cause of cancer death in the US.

Dr. Storz says there is a pressing need for new treatments and ways to prevent pancreatic cancer, one of the most aggressive human cancers. The disease is the fourth leading cause of cancer death in the US.

On average, patients with pancreatic cancer face a 20% chance of living more than 1 year after diagnosis. The main reason for such a poor prospect is that symptoms do not show until the cancer is well advanced.

According to the National Cancer Institute, in 2014, over 46,400 people will be diagnosed with pancreatic cancer and over 39,500 will die of the disease.

For the study, the team investigated cells containing mutations of a gene that regulates a cell division protein. Called Kras, the gene is known to be mutated in over 95% of pancreatic cancer cases.

Study details steps that mutated cells follow as they transform into pre-cancerous lesions

The researchers describe how they identified the detailed steps that Kras-mutated acinar cells follow as they change into duct-like cells with properties similar to stem cells – a type of cell often found in cancer.

They noticed that Kras proteins in the acinar cells switched on a molecule called ICAM-1. This in turn attracts macrophages – inflammatory immune cells that release a variety of proteins, including some that loosen the pancreatic cell structures. When they become looser, the acinar cells then transform into different types of cell – including the type that leads to pre-cancerous lesions.

Dr. Storz says their study shows a “direct link between Kras mutations and the inflammatory environment that drive the initiation of pancreatic cancer.”

Team found two ways to stop process toward pre-cancerous lesions in mice

The team also found they could halt the process in mice. They did this in two ways. One way was by depleting the macrophages, and the other way was by blocking the transforming cells with an antibody that shuts down ICAM-1.

“Doing either one reduced the number of precancerous lesions,” says Dr. Storz, noting that an antibody that blocks ICAM-1 has already been developed and is currently being tested for treating stroke, rheumatoidarthritis, and other ailments.

He also says the key to developing targeted ways to prevent and treat pancreatic cancer will be understanding “the crosstalk between acinar cells with Kras mutations and the microenvironment of those cells.”

The National Institutes of Health funded the research.

The study follows another recent report from Medical News Today about the prospect of a simple blood test for pancreatic cancer, as more potential biomarkers for the disease emerge.

Why Sugar is Toxic to Your Health .

Why Sugar is Toxic to Your Health


Why is sugar toxic to the body? In 1957, Dr William Coda Martin attempted to answer the question: When is food a food and when is it a poison?

His definition of “poison” was: “Medically: Any substance applied to the body, ingested or developed within the body, which causes or may cause disease.” Dr. Martin determined that sugar is indeed a poison and thus, classified it as such.

Sugar’s toxic wrath is in part due to it’s composition as a purely refined carbohydrate. It is a chemical. Our bodies cannot utilize this poison because it has been stripped of all vitamins, minerals, and all other nutrients that help your body create optimum health. What occurs when sugar is ingested is a balancing act that takes place inside our bodies.

Because balance or homeostasis is so essential to our bodies, the minerals sodium, potassium, magnesium and calcium are utilized to create this balance. For example, in order to neutralize the blood from sugar (sugar creates an acidic body), calcium is taken from the bones and teeth, thus, decay and osteoporosis may occur. If sugar is consumed every day, our bodies eventually will be depleted of these very important minerals and leave you with mineral deficiencies that depletes your health and weakens your defenses against disease. This process allows the dreaded free-radicals to cause major damage. Think of free-radicals as your body’s equivalent to nuclear destruction.

Excess sugar affects every single organ in the body – including the liver. The liver is where sugar is stored in the form of glucose. Eventually, if sugar is consumed every day, your liver will be unable to store the excess sugar and is returned to the blood in the form of fatty acids. Guess where those fatty acids are stored? They are stored in the most inactive areas: The belly, the buttocks, the breasts and the thighs. After these areas have been filled-up, the fatty acids then get distributed to organs such as the heart and kidneys.

Sugar hinders the body’s immune system and predisposes people to illness and disease. Our white blood cells are adversely affected. Here are just some health conditions associated with over-consumption of sugar: Obesity, cancer, aging, cardiovascular disease, high blood pressure, adult-onset diabetes, eczema, kidney stones, depression, candida (yeast over-growth), anxiety, dental cavities, atherosclerosis, poor brain function and countless other conditions.

Cancer is affected by sugar due to the cancer cell’s main source of fuel: Glucose. By controlling blood glucose, the cancer cells are starved for fuel and the immune system is bolstered.

We think of sugar as the granulated white stuff but it can be disguised in many forms such as: corn syrup, fructose, sucrose, lactose, molasses, honey, maltose, fruit juice and many other forms. It is essential that we become aware of the hidden sources of sugar found in processed foods.

In order to create optimum health, it is essential that we mindfully choose whole grains, fruits, vegetables, lower our stress levels, exercise and AVOID SUGAR.

To Your Good Health!

Estrogen May Protect Heart by Inhibiting Mineralocorticoid

Estrogen inhibition of a key receptor that helps regulate blood pressure may be a new mechanism by which premenopausal women are protected from cardiovascular disease, researchers reported.

Findings from in vitro investigations published in the November issue of the journal Endocrinology suggest that estrogen signaling through the estrogen receptor protects the circulatory system by inhibiting the detrimental effects of the steroid hormone aldosterone signaling through the mineralocorticoid receptor.

Aldosterone activation of the mineralocorticoid receptor (MR) in the kidneys regulates sodium and fluid balance. MR also is expressed in the vasculature and in many other tissues. The steroid hormone has been shown to contribute to cardiovascular disease in both animal models and humans, researcher Iris Jaffe, MD, PhD, and colleagues from Tufts University, Boston, wrote.

It is well known that premenopausal women have increased protection against myocardial infarction and cardiovascular mortality compared to men of the same age, and endogenous estrogen is widely believed to mediate this protective effect. But the mechanisms for this have not been well understood, Jaffe explained in an interview.

“In clinical trials when we give estrogen back to postmenopausal women, we don’t always see the same benefits,” she told MedPage Today. “If we have a better understanding of how (endogenous) estrogen benefits women we might be able to mimic that.”

Mineralocorticoid Damages Blood Vessels

Jaffe has been studying the mineralocorticoid receptor in her lab at Tufts’ Sackler School of Graduate Biomedical Sciences for close to a decade.

“The mineralocorticoid receptor is a well known regulator of blood pressure, but in the last decade it has also become clear that mineralocorticoid receptors also damage blood vessels and promote heart attacks and strokes,” she said.

Study co-author Richard Karas, MD, PhD, studies the effects of estrogen on the cardiovascular system, and Jaffe said whenever mineralocorticoid receptor was found to have a detrimental effect on the vasculature of cells in her lab, Karas’ lab would find that estrogen prevented the same effect.

“We got to thinking that maybe these two receptors work together, and that is what led to this study,” Jaffe said.

The researchers hypothesized that estrogen (E2) signaling through the estrogen receptor (ER) may protect the vasculature by inhibiting the detrimental effects of aldosterone (aldo) signaling through the MR.

In a series of in vitro studies they demonstrated that E2-activated ER inhibited MR-mediated gene transcription from the mouse mammary tumor virus reporter in human embryonic kidney-293 cells. In contrast, aldo-activated MR did not affect ER-mediated gene transcription.

“The ER N terminus (amino acids 1–253) containing part of the DNA-binding domain is sufficient to inhibit MR genomic function, although point mutations reveal that DNA binding, ligand-independent activation, and rapid nongenomic ER signaling are not required for this effect,” the researchers wrote. “Furthermore, ER and MR are part of a complex in cell lysates, with amino acids 1–233 of the ERα N terminus being sufficient to complex with the MR.”

The ability of ER to inhibit MR-mediated gene transcription was also found to correlate with the ability of ER segments to both localize to the nucleus and complex with the MR. In cultured vascular endothelial cells expressing ER, E2 inhibited aldosterone induction of the vascular MR target gene intercellular adhesion molecule-1 (ICAM-1).

ICAM-1 induction by endothelial MR is known to promote vascular inflammation that could contribute to the mechanism of aldo-induced atherosclerosis. E2 also inhibits aldo induction of ICAM-1 protein and prevents aldo-enhanced leukocyte adhesion to endothelial cells.

‘Studies Support New Model for Cardio Protection’

The in vitro studies support a new model in which E2-activated ER in endothelial cells forms a complex with mineralocorticoid receptor in the nucleus to modulate mineralocorticoid regulation of the proinflammatory gene ICAM-1, the researchers noted.

“Although further studies are needed to clarify the detailed molecular mechanism(s) by which ER abrogates MR-mediated gene regulation, our findings are consistent with several possible mechanisms,” they wrote. “First, the data support that the mechanism requires ERα to form a complex with MR in the nucleus, either through direct binding or indirectly via other proteins, and that this interaction blocks MR function as a transcriptional factor.”

However, even though the ER segment 1–253 was identified as the smallest portion able to inhibit MR transcriptional activity, the smaller segment 1–233 was able to complex with MR.

“The explanation for this discrepancy may come from our finding that nuclear localization of 1–233 was much weaker than 1–253, suggesting nuclear localization of ER may also be critical for its interference with MR-mediated transcriptional activity,” the researchers wrote. They added that “this mechanism appears to be distinct from the well-characterized dimerization interaction of the retinoid-X-receptor with non-steroid nuclear receptors including the thyroid, retinoid and vitamin D receptors because this does not require DNA binding by the ER.”

Jaffe and colleagues noted that once the ER forms a complex with MR in the nucleus, “multiple potential mechanisms are consistent with the data.”

“ER may prevent MR from interacting with a co-factor that is required for aldo-induced transcriptional activation but is not needed for ER transcriptional activation,” they wrote. “This is consistent with the finding that MR does not mutually inhibit transcriptional activation of ER. Recently posttranslational modification of the MR by phosphorylation has been shown to modulate MR transcriptional function in specific renal cell types. Thus, another possibility could be that complexing with ER in the nucleus facilitates MR modifications that alter its transcriptional function.”

The researchers cited several potentially important study limitations including the use of a transformed human embryonic kidney cell line (HEK293) with overexpressed ER and MR proteins and a transfected supercoiled plasmid containing the MMTV, MR-responsive element as a transcriptional reporter for their initial studies exploring interactions between ER and MR transcriptional function.

“This system, although highly artificial, was chosen because these HEK293 cells lack endogenous ER, MR and GR, and hence, the expression of receptors and ER mutants could be carefully controlled to specifically and independently examine transcriptional function and the ER and the MR,” they wrote.

The lack of known vascular MR target gene DNA binding sites and an adequate MR antibody for chromatin immunoprecipitation prevented the use of more modern methods to confirm endogenous MR binding.

In Vivo Studies Needed to Confirm Findings

The researchers also noted that since ER expression is rapidly down-regulated in cultured cells, the ER in the studies was exogenously expressed.

“Future studies in whole vessels or in vivo will be needed to interrogate this novel mechanism in the setting of physiological levels or MR and ER,” they wrote.

Jaffe said she hopes next to study the estrogen receptor-mineralocorticoid receptor connection in a mouse model.

“If one of the benefits of estrogen is that it is preventing the detrimental effects of mineralocorticoid, we might be able to reproduce that effect in men and postmenopausal women by giving them mineralocorticoid antagonists,” she said.

Several anti-mineralocorticoid diuretics have been approved and are used to treat chronic heart failure, hypertension and a condition known as hyperaldosteronism.

“These drugs act on the mineralocorticoid receptor in the kidneys to lower blood pressure and decrease mortality in patients with heart failure, but I don’t think anyone has really thought about their blood vessel protective effect,” Jaffe said.

Action Points

  • Estrogen inhibition of a key receptor that helps regulate blood pressure may be a new mechanism by which premenopausal women are protected from cardiovascular disease.
  • Note that the in vitro studies support a new model in which estrogen-activated estrogen receptor in endothelial cells forms a complex with mineralocorticoid receptor in the nucleus to modulate mineralocorticoid regulation of the proinflammatory gene ICAM-1.

World’s Oldest Living People Have Their Genomes Sequenced

In the hopes of uncovering the genetic basis for extremely long life spans, scientists have sequenced the genomes of 17 of the world’s oldest living people.
Participants ranged in age from 110 to 116, and all but one were female. Many of these so-called “supercentenarians” were physically and cognitively fit into their old age — one participant practiced as a doctor until age 103, and another drove a car until age 107.
The ultimate goal of the research is to figure out how supercentenarians are able to “slow down the aging clock,” said study co-author Stuart Kim, a professor of developmental biology at Stanford University. If researchers are able to figure that out, they might be able to create a drug or vitamin that would do the same thing in non-superagers, so that people could extend their “middle age” for many years, Kim said.

None of the supercentenarians in the study had heart disease, stroke or diabetes — diseases that are very common in old age — and just one participant had been diagnosed with cancer. In contrast, in the United States, about half of people have been diagnosed with cancer by age 85, and 35 percent have been diagnosed with heart disease.
Unfortunately, the secret to a long life span remains a mystery for now — a first analysis of the genomes did not reveal any rare genetic mutations that might have been responsible for the participants’ extraordinary ages.
However, the researchers have made the genome sequences publically available in the hope that future research might discover secrets to their long life.
“The best way forward is for people to pool their data so we can compare all the supercentenarians,” Kim said.
Previous studies have found certain variations in the genetic code of centenarians. But these variations, are also relatively common in the general population — for example, they might be found in 10 percent of people.
In the new study, the researchers hypothesized that people who live to be 110 or older may share a rare mutation, or a rare gene, that would be responsible for their long life, that was not common in the rest of the population.
The study was not able to find such a gene, but this may have been because there were too few people in the study to detect meaningful differences.
In addition, the genetic basis of life span is likely a complex trait — it could be that many small differences across a person’s genome combine together to make for a long life span, Kim said. Or, it could be that, although there is no shared gene that is common among supercentenarians, individual families may each have their own gene for longevity, said Kim, who conducted the work with Stephen Coles, of the Gerontology Research Group in Los Angeles, Leroy Hood of the Institute for Systems Biology in Seattle, and colleagues.

Man creates font designed to help dyslexics read

A font created by a dyslexic designer could potentially bring relief to the up to 20% of the U.S. population who experience symptoms of dyslexia.


The typeface, called Dyslexie, better distinguishes letters that are often confused by dyslexics and slows them down while reading. Developed in 2008 and first released in English in 2011, the custom font is receiving renewed attention while on display at the Istanbul Design Biennial, The Guardian reports.

According to his website, Boer developed Dyslexie as a thesis project at the Utrecht Academy of Art. Subsequent studies at the University of Twente and the University of Amsterdam yielded impressive results — 84.3% of dyslexic children and adults who participated in the study could read Dyslexie faster than other typefaces, with 77.7% fewer mistakes.

The Mayo Clinic defines dyslexia as “a learning disorder characterized by difficulty reading due to problems identifying speech sounds and learning how they relate to letters and words.”

Dyslexie is one of several fonts that help those with dyslexia. Others include OpenDyslexic and Spellex DysLex. Dyslexie is available as a free browser extensionhere.

Flu vaccines are NOT safe for pregnant women, warns doctor

Award-winning Dr. David Brownstein, a board-certified family physician and medical director of the Center for Holistic Medicine in West Bloomfield, MI, has a warning for pregnant women and those considering becoming pregnant. In a nutshell, he says that if you’re thinking about getting a flu vaccine, don’t.(1,2)

His words are ones that are in direct opposition to a recently published Committee Opinion paper by the American Academy of Obstetrics and Gynecology (ACOG), where the organization states that women during this time in their life should get a flu shot. AGOG notes that doing so is “essential” due to the increased risk that a pregnant woman faces when it comes to pandemic and seasonal influenza.

“It is particularly important,” AGOG mentions, “that women who are or will be pregnant during influenza season receive an inactivated influenza vaccine as soon as it is available. They continue to express a view that is steeped with a sense of urgency, stating, “It is imperative that obstetrician-gynecologists, other health care providers, health care organizations, and public health officials continue efforts to improve the rate of influenza vaccination among pregnant women.”(1)

Imperative? Not so fast, Dr. Brownstein warns.

Flu vaccine safety maintained despite lack of studies

He points out that the flu vaccine is preserved with mercury, a known neurotoxin, in the form of thimerosal. Thimerosal, which apparently contains mercury and is used to keep the flu vaccine free of fungi and bacteria, has been banned in other countries outside of the United States for over 20 years since it’s been linked with causing nervous system injury, coma and death.(1,3)

Still, ACOG maintains that it’s safe for pregnant women or those considering becoming pregnant to have a flu shot, claiming that no scientific claims prove that the vaccine can be harmful to children. Well, of course they can hold tight to such a statement; Dr. Brownstein says that the ACOG can say this because studies haven’t been conducted that consider the effects of thimerosal injection into a pregnant women and its harm to the fetus. In other words, there can’t be a scientific claim about something if a study about it hasn’t been done in the first place, right? By omitting key details like studies not even having been conducted, the public is severely mislead. Furthermore, Dr. Brownstein maintains that there aren’t even studies proving that injection of thimerosal is safe for a pregnant woman.(1)

However, several animal studies have found that injected thimerosal does indeed cross the blood-brain barrier, affecting brains of injected animals as well as their fetuses.(1)

Common sense is imperative; flu vaccine isn’t

One only needs to use common sense, he says, to know that injecting toxicity into the body isn’t something that’s going to put a woman and her soon-to-be-born baby, or anyone for that matter, in the best of health. In fact, the Department of Defense classifies mercury as a material that’s so harmful that inhalation, ingestion or absorption through skin could lead to death.(3)

Even the package insert (PI) for the FluLaval quadrivalent vaccine states, “There are… no adequate and well-controlled studies in pregnant women and FluLaval quadrivalent should be given to a pregnant woman only if clearly needed.”(1)

His commonsense reasoning echoes what Natural News reports have also warned about. Quite simply, stay far away from the flu vaccine, pregnant or not.

Consider the recent news that, just four days after receiving a flu shot, 10-year-old Marysue Grivna of Tampa, Florida, now has a brain disease called acute disseminated encephalomyelitis (ADEM). The disease attacks protective parts of the brain designed to keep the spinal cord and brain functioning properly and behaves in a way similar to multiple sclerosis. She’s now nonverbal, hospital bed- and wheelchair-bound and primarily eating with the assistance of a tube.(4)







Can’t dance? There might be a scientific explanation .

Image: Taylor Swift, Shake It Off, Big Machine Records
Can’t dance? There might be a scientific explanation

Researchers have discovered why some people can’t keep a beat or tap along with music – they could have a rare condition known as ‘beat-deafness.’

If you look more like an awkward robot than Kevin Bacon in Footloose when on the dancefloor, don’t feel bad – there may be a scientific explanation for your inability to dance. New research suggests that you could be ‘beat-deaf’ – which means you’re simply incapable of moving to the rhythm of music.

To investigate if there was a biological excuse for all those clumsy dancers out there, researchers from McGill University and the University of Montreal in Canada compared two people with suspected beat-deafness to 32 control participants. The participants were asked to tap their feet evenly without any sound, which they all managed to do easily. This ruled out a general motor deficit. But when music was added, the beat-deaf participants weren’t able to keep in sync with the time changes, the team reports in the journal The Philosophical Transactions of the Royal Society.

The team concluded that beat-deafness, though very rare, is a disorder that affects how the body’s internal biological rhythms change to react to external cues. Biological rhythms are the natural patterns that allow us to perform activities such as eating and sleeping. Other rhythmic behaviours are driven by external cues, such as when we moderate our walking speed to match our partner’s.

The findings suggest that beat-deaf individuals are less able to synchronise their biological rhythms with an external cue, which could explain why they find it difficult to tap their feet or dance to music.

“While most people can adapt their rhythms in response to an external cue, some people are less able to do that”, said Caroline Palmer, psychologist and lead researcher, in a press release. “We tested what makes beat-deaf individuals different, by seeing how people whose biological rhythms may not respond normally to external cues adapt to an external beat.”

In a separate study published last week, scientists explored the link between a child’s ability to assess a musical rhythm and master various grammar exercises. The results revealed that children who performed well in one skill also performed better than average in the other skill, suggesting that music could benefit a child’s learning ability.

Many studies have shown the benefits of understanding rhythm in other aspects of our life, such as multi-tasking and academic performance. It’s fascinating that most of us evolved to have an innate biological clock which programs responses to external rhythms, but what does it mean for those who are beat-deaf?

While being rhythmic is a great talent to have, for all the rhythm-less people out there, remember that there’s scientific explanation to justify your bizarre dance moves.

Scientists Photograph Soul Leaving Body At Death, According To New Study

I recently came across an article that I thought was interesting enough to share, and I think I may be able to provide proper scientific support for these claims for the very first time ever by presenting the original published study.  The timing of astral disembodiment in which the spirit leaves the body has allegedly been captured by Russian scientist Konstantin Korotkov, who photographed a person at the moment of his death with a bioelectrographic camera.

According to Korotkov, navel and head are the parties who first lose their life force (which would be the soul) and the groin and the heart are the last areas where the spirit before surfing the phantasmagoria of the infinite.

In other cases, according to Korotkov, the “the soul” of people who suffer a violent and unexpected death usually manifests a state of confusion in your power settings and return to the body in the days following death. This could be due to a surplus of unused energy.

The technique developed by Korotkov, who is director of the Research Institute of Physical Culture, St. Petersburg, is endorsed as a medical technology by the Ministry of Health of Russia and is used by more than 300 doctors in the world for stress and monitoring progress of patients treated for diseases such as cancer. Korotkov says his energy imaging technique could be used to watch all kinds of imbalances biophysical and diagnose in real time and also to show if a person does have psychic powers or is a fraud.

This technique, which measures real-time and stimulated radiation is amplified by the electromagnetic field is a more advanced version of the technology developed for measuring Semyon Kirlian aura.  Here is an interview with Dr. Korotkov talking more about his practice:

Now, before we get into the good stuff, let’s consider a few things.

Dr. Korotkov is the author of more than 200 papers in leading journals on physics and biology, and he holds 17 patents on biophysics inventions. Prof. Korotkov has led a research career for over 30 years, combining rigorous scientific method with an insatiable curiosity about things of the spirit and the soul with deep respect for all life. He is also a scholar in philosophy and a serious mountaineer of 25 years experience. He has given lectures, seminars and training sessions in 43 countries, presenting papers and workshops at more than 100 national and international conferences.

dr k

If one were to question the credibility of this story, it could not be on the basis that the scientist is not credible.  He is the Professor of Computer Science and Biophysics at Saint-Petersburg Federal University of Informational Technologies, Mechanics and Optics.  Professor of Research in Saint Petersburg Academy of Physical Culture, and the President of the International Union for Medical and Applied Bioelectrography just to name a few.

I visited his website, and looked through the articles he has published.  He has tons of papers on measuring human energy fields, the scientific measurement of the human aura, and other fascinating topics.  But upon my research, I realized I stumbled upon the original paper which represents a summary of results of experimental research on post mortem evoked bioelectrographic activity of the human body on the basis of Kirlian effect.  The study titled “Experimental investigations of post mortem bioelectrographic images of human fingers.” can be found HERE.

They were measuring the left over energy field of dead human bodies using GDV technology.   Once an hour, they would go down to the experimental room, dry a photo-paper list (date, time), put the photo-paper under the fingers and take two gas-discharge photos onto one list, having simultaneously exposed a metal bench mark object on the same list.  On the whole, in the period of two years 26 experimental sessions were put into
practice, each of which took from three to five days. Among the dead were both men and
women aged from 19 to 70.  The found that there were 3 main groups of photographs that emerged during their experiments, and these photographs were always correlated with the type of death that person endured:

1) Curves on the photo readings with relatively small variation amplitude.  This was associated with a “calm death” as a result of natural motives, stipulated by organism’s condition, generally in the old age.

2) Curves on the photo readings with availability of one pronounced peak for several hours after death and
relatively small amplitude afterwards.  This was associated with an “unexpected death” in the issue of traffic accident followed by craniocerebral trauma.

3) Curves on the photo readings large amplitude variations, which continue for a long time.  These were associated with an “unnatural death” as a consequence of unfavourable concourse of  circumstances: a suicide, a murder, improper medical care, lungs clot.

They concluded that these are soft or left-over energetic substances of the dead that remained with the corpse and can be photographed, with the photos correlating with the time of trauma they experiences.   There exists a sort of fingerprint of the soul that can be detected within the body.  Here is a photo showing the nature of GDV photography:


They say that the study gives credence to the idea of our physical systems being correlated with our energy-informational structures (souls). “We are  talking about energy-informational structure as an objective space-field structure, interconnected with human body, but existing independently of it, particularly for some
definite time after death. This structure originates from the birth and changes in the process of morphogenetic synergism.”

As I quote from the original paper which can be found here:

“We have  received provisional data demonstrating that in some cases in a few days before death the raise of patient’s activity and increase of gas-discharge intensity are observed. Such data may provide for a new information on the process of Transition to other Life.”

So for the first time ever, the infamous case finally has the original study coupled with it to strengthen its claims.  The photos of the soul leaving the body at death would look nothing like the one at the top of the article, and would look more like spikes and amplitudes in GDV energetic photographs that correlate with the nature of the persons death and the time at which they died.  There may be other similar studies among the hundred he has published which have yet to be dug up and reported upon, but I think that it’s interesting to at least acknowledge the fact that photographs of our energy-informational systems can be objectively photographed within our bodies after death.


Speaking More Than One Language Could Sharpen Your Brain

Skip the sudoku, try learning French

Speaking more than one language does the brain some good.

A recent study found that bilingual speakers may actually process information more efficiently than single-language speakers. Researchers from Northwestern University, in Illinois, and the University of Houston used brain imaging to look at bilingual people’s comprehension abilities. They found that people who speak more than one language are comparatively better at filtering out unnecessary words than monolinguals, whose brains showed that they had to work harder to complete the same mental tasks.

The study, published in the journal Brain and Language, used functional magnetic resonance imaging (fMRI) to look at what’s called coactivation and inhibition in the brain. Coactivation is the ability to have both languages simultaneously active in the brain, while inhibition is that ability to select a correct language while hearing more than one at a time. The researchers studied 17 Spanish-English bilinguals and 18 monolinguals, and had them undergo tests that assessed their brains’ ability to eliminate irrelevant words.

For example, in one task the participants heard the word cloud and were then immediately shown four pictures. One of the photos was of a cloud, and another was a similar-sounding word like clown. The goal was to watch how quickly the brain could make connections to the correct word. Bilinguals were consistently better at the task.

The results create a bit of a chicken-or-the-egg scenario. Is a bilingual person better at such tasks because of their expertise in both languages, or are people with greater comprehension capacity better equipped to master multiple languages? It could be a mixture of the two. The researchers of the new study believe that being bilingual is a constant brain exercise. So instead of tackling a puzzle, why not give a new language a shot, if not solely for the brain challenge.

Gut–brain link grabs neuroscientists .


Feeding mice the bacterium Bacteroides fragilis can reverse autism-like symptoms.

Companies selling ‘probiotic’ foods have long claimed that cultivating the right gut bacteria can benefit mental well-being, but neuroscientists have generally been sceptical. Now there is hard evidence linking conditions such as autism and depression to the gut’s microbial residents, known as the microbiome. And neuroscientists are taking notice — not just of the clinical implications but also of what the link could mean for experimental design.

“The field is going to another level of sophistication,” says Sarkis Mazmanian, a microbiologist at the California Institute of Technology in Pasadena. “Hopefully this will shift this image that there’s too much commercial interest and data from too few labs.”

This year, the US National Institute of Mental Health spent more than US$1 million on a new research programme aimed at the microbiome–brain connection. And on 19 November, neuroscientists will present evidence for the link in a symposium at the annual Society for Neuroscience meeting in Washington DC called ‘Gut Microbes and the Brain: Paradigm Shift in Neuroscience’.

Although correlations have been noted between the composition of the gut microbiome and behavioural conditions, especially autism1, neuroscientists are only now starting to understand how gut bacteria may influence the brain. The immune system almost certainly plays a part, Mazmanian says, as does the vagus nerve, which connects the brain to the digestive tract. Bacterial waste products can also influence the brain — for example, at least two types of intestinal bacterium produce the neurotransmitter γ-aminobutyric acid (GABA)2.

The microbiome is likely to have its greatest impact on the brain early in life, says pharmacologist John Cryan at University College Cork in Ireland. In a study to be presented at the neuroscience meeting, his group found that mice born by caesarean section, which hosted different microbes from mice born vaginally, were significantly more anxious and had symptoms of depression. The animals’ inability to pick up their mothers’ vaginal microbes during birth — the first bacteria that they would normally encounter — may cause lifelong changes in mental health, he says.

Similarly, a 2013 study from Mazmanian’s lab found that a mouse model with some features of autism had much lower levels of a common gut bacterium called Bacteroides fragilis than did normal mice3. The animals were also stressed, antisocial and had gastrointestinal symptoms often seen in autism. Feeding B. fragilis to the mice reversed the symptoms. The group also found that the mice with these symptoms had higher levels of a bacterial metabolite called 4-ethylphenylsulphate (4EPS) in their blood, and that injecting that chemical into normal mice caused the same behavioural problems.

Yet even those at the forefront of the research remain sceptical that the findings will translate into treatments for humans. The evidence that probiotics affect human behaviour “is minimal to say the least”, Mazmanian acknowledges. Still, he says, a growing number of researchers are starting to look at some mental illnesses through a microbial lens.

There are implications for basic research too. In another study to be presented at the meeting, veterinarian Catherine Hagan at the University of Missouri in Columbia compared the gut bacteria in laboratory mice of the same genetic strain that had been bought from different vendors. Their commensals differed widely, she found: mice from the Jackson Laboratory in Bar Harbor, Maine, for instance, had fewer bacterial types in their guts than did mice from Harlan Laboratories, which is headquartered in Indianapolis, Indiana.

Such differences could present a major complication for researchers seeking to reproduce another lab’s behavioural experiments, Hagan says. When her team transplanted bacteria from female Harlan mice into female Jackson mice, the animals became less anxious and had lower levels of stress-related chemicals in their blood. Hagan notes that when a lab makes a mouse by in vitrofertilization, the animal will pick up microbes from its surrogate mother, which might differ greatly from those of its genetic mother. “If we’re going to kill animals for research, we want to make sure they’re modelling what we think they’re modelling,” she says.

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