Effect of Fluconazole Prophylaxis on Candidiasis and Mortality in Premature InfantsA Randomized Clinical TrialFluconazole Prophylaxis, Candidiasis, and MortalityFluconazole Prophylaxis, Candidiasis, and Mortality

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Invasive candidiasis is associated with impaired neurodevelopment and mortality in premature infants. Benjamin and colleagues assessed the effect of fluconazole prophylaxis (twice weekly for 42 days) in a multicenter, randomized, placebo-controlled trial involving 361 premature infants with birth weight less than 750 g. The authors report that compared with placebo, fluconazole prophylaxis did not result in a lower incidence of a composite outcome of death or invasive candidiasis.

Controversy exists as to whether use of steroids after hepatoportoenterostomy improves clinical outcomes in infants with biliary atresia. To explore this question, Bezerra and colleagues randomly assigned 140 infants with biliary atresia to receive high-dose steroid therapy or placebo following hepatoportoenterostomy. The authors report steroid treatment did not result in a statistically significant improvement in biliary drainage 6 months after surgery.

Munoz and colleagues assessed the safety and immunogenicity of tetanus toxoid, reduced diphtheria toxoid, and acellular pertussis (Tdap) immunization during pregnancy in a randomized, placebo-controlled study involving 48 pregnant women. The authors found no increased risk of adverse events among the women who received antepartum Tdap or their infants; high levels of pertussis antibodies in the infants; and no significant alteration in infant response to routine DTaP vaccination. In an Editorial, Jiménez-Truque and Edwards discuss maternal pertussis immunization to protect young infants.

In an analysis of longitudinal data from more than 2 million Swedish children—including identification of twin, sibling, and cousin pairs—Sandin and colleagues assessed the familial aggregation of autism spectrum disorders. The authors found that the individual risk of autism spectrum disorder and autistic disorder increased with increasing genetic relatedness, with an estimated heritability of approximately 50%. In an Editorial, Schendel and colleagues discuss genetic and environmental contributions to autism spectrum disorders.


To examine trends in prevalence of type 1 and type 2 diabetes among youth, Dabelea and colleagues analyzed medical record data from more than 3 million youth from birth to 19 years in 5 areas of the United States. The authors report that between 2001 and 2009 the prevalence of both type 1 diabetes and type 2 diabetes increased.

UK child mortality rate one of the highest in Western Europe .


AFP Photo/Gabriel Bouys

AFP Photo/Gabriel Bouys

Britain has one of the highest mortality rates in Western Europe for children under five, new research has revealed. Experts say factors like poverty, deprivation and smoking during pregnancy contributed to the premature deaths of 3,000 children in 2012.

Infants born in Great Britain are more likely to die before their fifth birthday than any other country in Western Europe, apart from Malta, according to a study by the University of Washington and published in the Lancet journal.

The study calculates the mortality rate at 4.9 deaths for every 1,000 births in the UK, which is 25 percent higher than the Western European average. The study’s authors said they were surprised that a developed country that had pioneered a public health system had higher rates than poorer countries like Greece and Cyprus.

The UK’s mortality rate is comparable with those of Poland and Serbia.

“We were surprised by these findings because the UK has made so many significant advances in public health over the years,” Dr Christopher Murray, the study’s principal director told The Guardian. “The higher than expected child death rates in the UK are a reminder to all of us that, even as we are seeing child mortality decline worldwide, countries need to examine what they are doing to make sure more children grow into adulthood.”

The study highlights the fact that although the childhood death rate fell between 1990 and 2013, the speed of the drop has slowed recently.

Poverty and deprivation caused by cuts in welfare contribute to the large amount of deaths among children under five in the UK, suggests the study. Infants are more likely to die if they come from a poorer family, or have parents that smoked and drank during pregnancy.

Editor-in-chief of the Lancet, Richard Hooton said that one of the main factors contributing to the high rate of mortality was “the poor organization of children’s health services in the UK.”

“Until our politicians begin to take the health of children – the health of the next generation of British citizens – more seriously, newborns and older children will continue to suffer and die needlessly,” he told the Times.

The countries with the least deaths of infants under five in Europe are Iceland, Andorra and Sweden. In addition, countries that surpassed Britain outside Europe included Australia, Israel, Japan, Singapore and South Korea.

The UK has been hit hard by the financial crisis in Europe and as a result has seen a rise in poverty and the use of food banks across the country. According to The Trussell Trust, Britain’s largest food bank charity, 913,138 people received emergency food aid from the organization in 2013-2014, compared to just 346,992 in 2012-2013 – marking an increase of 163 percent. The organization has said the coalition government’s harsh cuts to Britain’s welfare system have had a significant effect on the poorer population.

Atypical Form of Alzheimer’s Disease May be Present in a More Widespread Number of Patients, Mayo Clinic Says | Mayo Clinic News Network | 8133725

Neuroscientists at Mayo Clinic in Florida have defined a subtype of Alzheimer’s disease (AD) that they say is neither well recognized nor treated appropriately.

The variant, called hippocampal sparing AD, made up 11 percent of the 1,821 AD-confirmed brains examined by Mayo Clinic researchers — suggesting this subtype is relatively widespread in the general population. The Alzheimer’s Association estimates that 5.2 million Americans are living with AD. And with nearly half of hippocampal sparing AD patients being misdiagnosed, this could mean that well over 600,000 Americans make up this AD variant, researchers say.

In an oral presentation at the annual meeting of the American Academy of Neurology in Philadelphia, scientists say hippocampal sparing AD often produces symptoms that are substantially different from the most commonly known form of AD, which affects the hippocampus, the center of memory.

The patients, mostly male, are afflicted at a much younger age, and their symptoms can be bizarre — behavioral problems such as frequent and sometimes profane angry outbursts, feelings that their limbs do not belong to them and are controlled by an “alien” unidentifiable force, or visual disturbances in the absence of eye problems, researchers say.

They also decline at a much faster rate than do patients with the most common form of AD.

“Many of these patients, however, have memories that are near normal, so clinicians often misdiagnose them with a variety of conditions that do not match the underlying neuropathology,” says the study’s lead author, Melissa Murray, Ph.D., an assistant professor of neuroscience at Mayo Clinic in Florida.

Many of these patients are diagnosed with frontotemporal dementia, a disorder characterized by changes in personality and social behavior, or corticobasal syndrome, characterized by movement disorders and cognitive dysfunction. Language dysfunction is also more common in hippocampal sparing AD, although patients do not have vocal or hearing deficits.

“What is tragic is that these patients are commonly misdiagnosed and we have new evidence that suggests drugs now on the market for AD could work best in these hippocampal sparing patients — possibly better than they work in the common form of the disease,” Dr. Murray says.

The researchers benefit greatly from one of the largest brain banks in the country — more than 6,500 brain donations — as well as a collaborative environment between neuroscience research and neurology at Mayo Clinic, she says.

Both hallmark proteins of AD — amyloid beta (Aβ), which forms Aβ plaques, and tau, which produces tangles — are found across all subtypes of AD, including hippocampal sparing AD. The researchers developed a mathematical algorithm to classify AD subtypes using tangle counts. “What is fascinating is that all the AD patient subtypes had the same amount of amyloid, but for some reason tau tangles were found in strategic cortical regions disproportionate to the hippocampus.”

In these patients, tau preferentially damages and eventually destroys neurons in parts of the brain involved in behavior, motor awareness and recognition, as well as use of speech and vision, Dr. Murray says.

She says she hopes this research, the second high-profile Mayo study to highlight hippocampal sparing AD, will “open the minds” of clinicians who are trying to diagnose dementia, helping them understand that loss of memory is not present in every AD patient.

“Our studies support the notion that dementia related to AD does not necessarily equate to a loss of memory, and points to the need for more research in amyloid and tau imaging biomarkers to help clinicians accurately diagnose AD — regardless of subtype,” Dr. Murray says.

Asthma morbidity linked to higher urinary dichlorophenol level.

Asthma morbidity in patients with atopy and a history of wheezing is associated with high urinary dichlorophenol levels which are also associated with greater total serum IgE, according to researchers.

“The present results indicate that atopic wheezers with urinary dichlorophenol levels in the upper tertile are more likely to have physician-diagnosed asthma, to miss days from work or school because of wheezing, to require medications for wheezing, or to have wheezing with exercise,” researchers wrote.

Elina Jerschow, MD, MSc, of the Albert Einstein College of Medicine/Montefiore Medical Center in Bronx, NY, and colleagues collected data from a sample of 2,125 participants aged at least 6 years from the US National Health and Nutrition Examination Survey 2005–2006. They identified a subsample of patients who reported wheezing in the past year (n=250).

The subsample was broken down into the following categories: atopic or nonatopic wheezers with higher 2, 5–dichlorophenol levels who were more frequently diagnosed with asthma by a physician (OR=4.7 for highest vs. the lowest tertile, P<.001), required more prescriptions for asthma medications (OR=2.2, P=.046), and reported more exercise-induced wheezing (OR=5.8, P=.045) vs. atopic wheezers with low dichlorophenol levels, according to data.

Jerschow and colleagues also found that atopic wheezers with greater 2, 5– or 2, 4– levels were more likely to miss work (OR=10, P<.001) or school (OR=11.4, P<.01) because of wheezing.

Conversely, there were no significant associations between dichlorophenol levels and asthma morbidity measurements in nonatopic wheezers, researchers wrote. The 2–dichlorophenol metabolites appeared positively tied to increased serum IgE levels in the larger study sample, according to data.

“Prospective studies are necessary to determine the cause-and-effect relation underlying these associations in wheezers and those with asthma,” researchers concluded.

Why good memories are less likely to fade

Why good memories are less likely to fade http://www.bbc.co.uk/news/health-27193607

From the desk of Zedie.

Weight Loss Tips: Diet, Exercise, And Let Science Accelerate Your Metabolism For You

Popular culture likes to muddle the process of losing weight. In a lot of cases, it’s as simple (not easy, mind you) as eating right and occasionally exercising. For some people, however, no amount of weight loss tips can erase the fact their body is wired to be stubborn. A new study suggests overcoming this default setting may be possible.

Everyone’s cells take a different amount of time to complete their necessary reactions. This length of time is known as a person’s basal metabolic rate (BMR). A greater BMR means the cells are producing more energy, which ultimately means foods are digested faster and broken down for use — metabolized, basically. When the BMR diminishes, more energy is required to assist the cells’ poorer function to avoid unused calories being stored as fat. In the everyday world, this comes from the foods we eat and the exercise we get. Now science believes it can lend a hand.

The intervention involves a key protein known as (ahem) nicotinamide N-methyltransferase, or NNMT. The protein helps process vitamin B3 in the body and has been linked to certain types of cancer and Alzheimer’s disease. Scientists at Beth Israel Deaconess Medical Center have found an alternate use: regulating energy metabolism in fat tissue. When NNMT levels in mice were high, obesity and diabetes were common. When they were reduced, the mice grew leaner and lost their diabetes.

What ultimately makes this possible is the body’s futile cycle — a crisscrossing of opposing cycles that produces a net effect of zero, with the exception of heat lost in the form of energy. This is important because it means scientists can manipulate a subject’s futile cycle and produce more energy externally. When the research team reduced NNMT levels, a separate group of molecules, known as polyamines, revved up cellular reactions.

“What’s interesting about the polyamines is that the process of building and degrading them creates a biochemical cycle in which energy is used up,” explained co-author Dr. Daniel Kraus in a statement. “This is a futile cycle.”

If the team can translate its findings into human models, a treatment for obesity could be possible. Right now, nearly 35 percent of the American public is obese, according to the Centers for Disease Control and Prevention, and rates have been climbing for decades. As we put on weight, our metabolism slows. Poor diet and lack of exercise begin to mimic, and sometimes amplify, the effects of bad genetics.

At that point, hopping on the elliptical for a half hour may help, but the climb is much further uphill than it once was. And for the morbidly obese — people who can barely walk on their own, let alone jog around the block — anti-obesity treatments, such as liposuction and lap band surgery, become their only options. With the current research, surgery may not necessarily be the next step.

“Anti-obesity therapies could be of tremendous help, and NNMT looks to be a promising target for future therapeutic development,” said senior author Barbara Kahn. “Furthermore, because obesity is associated with an increased incidence of Alzheimer’s disease and certain cancers, disease states in which NNMT is also elevated” interventions may also “be beneficial in managing these other devastating conditions.”


Source: Kraus D, Yang Q, Kong D, et al. Nicotinamide N-methyltransferase knockdown protects against diet-induced obesity. Nature. 2014.

Facial recognition: is the technology taking away your identity? | Tech | The Guardian


From the desk of Zedie.

Do All Babies Look the Same? Depends on Who You Are.

I’ve heard the claim plenty of times from friends. “Why send out baby pictures?” one asked. “All newborns look exactly the same. They barely even look human.” Having a baby has made me more sensitive to the subtle differences in smushy-faced features, but the question remains: do babies actually look different? Can face-recognition tech work on tiny babies.

As it turns out, scientists have had these same questions, and found that if you’re a man, babies might look the same. Researchers at St. Andrews University showed pictures of babies to men and women of different ages. Can you pick out the cute one among the not-so-cute cohort? 

Cuteness is described by the researchers as: “protruding cheeks, a large forehead, and large eyes below the horizontal midline of the skull.” Turns out that men aren’t as astute at picking the cute baby.

The researchers surmise hormones rule women’s ability to notice the finer details of a newborn’s pudgy face. Women taking birth control pills, which raise hormone levels, were more likely to pick out the cute baby. Young and middle-aged women were able to pick out cute babies from bunch, while women aged 53-60 were just as unlikely to notice differences in the babies as men.

The researchers told a newspaper that their next studies would examine whether or not cuteness sensitivity is implicated in post-natal depression.

Is that my baby, all grown up?

Non-hormonally-charged people aren’t the only ones who have trouble distinguishing baby faces – computers also struggle to understand who’s a baby. When I added photos to Facebook recently, the website tagged my 7-month old as a middle-aged neighbor from Seattle. I don’t blame Facebook: I see that my little guy’s chubby cheeks and crinkly grin could be mistaken for adult faces.

So why are baby faces so difficult for computers to recognize? “There are a lot of shape changes between babies and adults, like nose changes, eyes shape, and mouth shape,” says Ira Kemelmacher-Schlizerman, a computer scientist at the University of Washington. With her colleagues, Kemelmacher-Schlizerman created a way to progressive age photos, starting with photos of babies just a few months old. Using thousands of photos of children and adults at many ages from the Internet, the researchers showed how to create average images from old sources.“We started from these photos since they were identified as the most challenging ones for current methods,” she says.

The new method was able to age real-life photos taken in all kinds of lighting, and this kind of work could help in recovering abducted children. Says Kemelmacher-Schlizerman, “It’s remarkable that we were able to achieve high quality results by applying average transformations estimated from internet photos. We didn’t include any craniofacial studies, change due to ethnicities, artificial addition of wrinkles, gray hair — all these can be added on top of the results we achieved automatically. The key idea of the work is to be able to automatically analyze large amounts of uncalibrated photos.” The research, funded by Google and Intel, led to a paper that will be presented at a Computer Vision and Pattern Recognition conference in Columbus, Ohio, this June.

Ira Kemelmacher-Shlizerman, http://grail.cs.washington.edu/aging/


Psychol Sci. 2009 Feb;20(2):149-54. doi: 10.1111/j.1467-9280.2009.02272.x. Epub 2009 Jan 17. The cutest little baby face: a hormonal link to sensitivity to cuteness in infant faces. Sprengelmeyer R1, Perrett DI, Fagan EC, Cornwell RE, Lobmaier JS, Sprengelmeyer A, Aasheim HB, Black IM, Cameron LM, Crow S, Milne N, Rhodes EC, Young AW.


Australian cardiologist regrows monkey hearts with human stem cells.

Dr James Chong says ‘very significant advance’ with macaque monkeys could soon be used to treat heart disease in humans


stem cells monkeys
Breakthrough research shows cardiac cells derived from human stem cells (green) meshed with monkey heart cells (red). Photograph: Murry Lab/University of Washington/PA

An Australian cardiologist has achieved what could prove a major breakthrough in treating heart disease, after working with US scientists to use human stem cells to regenerate the damaged hearts of monkeys.

A proof-of-principle study, published in Nature, showed that human embryonic stem cells boosted heart regeneration when transplanted into injured macaque hearts.

The findings represent the first time that scientists have been able to grow stem cell-derived heart muscle at a scale to treat large animals. The study was also successful in showing that the transplanted cells worked in tandem, rather than against, host tissue.

Previous work has shown that the human cardiac stem cells, known as cardiomyocytes, could be used on rodents. But the breakthrough with the macaque monkeys shows it could be feasible to use the same treatment in humans, albeit after several more years of research. More than 20,000 Australians die from heart failure each year.

Dr James Chong, a cardiologist at the Westmead Hospital and Sydney University lecturer, authored the research paper in collaboration with a University of Washington team, led by Charles Murry.

The researchers induced a heart attack in eight macaque monkeys by impeding the blood flow into the organ. Two weeks later, the stem cells were injected into the heart.

Over a period of three months, the hearts were shown to have regenerated dead tissue by up to 40% and achieved electromechanical coupling with the original host heart. However, it’s unclear from the study whether a full recovery would be possible via stem cell treatment.

Chong said the research was a “very significant advance” in the work to find better ways to treating heart failure.

“I think it’s realistic to assume that this could be used in humans sooner or later,” he told Guardian Australia. “A lot of work needs to be done but I don’t think we’re too far away.

“Heart failure is a growing problem. We’ve got better at treating heart attacks but it means that a lot of people are living longer with damaged heart muscle. This damage slowly spirals out of control and the medications we have only slow down the process.

“The only cure really is a heart transplant, which is obviously problematic. The human heart and brain are the least regenerative parts of the body.”

The researchers are now seeking further funding to advance the studies so that human clinical trials can eventually be undertaken.

Why Medicine Won’t Allow Cancer to Be Cured.

Imagine a commercial plane crashed and there were some fatalities involved. You can be sure that would make the headline of every major newspaper. Well, we have the equivalent of 8-10 planes crashing EVERY DAY with everyone on board dying from cancer.

Nearly two million Americans are diagnosed with cancer every year—one person out of three will be hit with a cancer diagnosis at some time in their lives, in spite of the massive technological advances over the past half-century.

Western medicine is no closer to finding a “cancer cure,” while cancer has grown into a worldwide epidemic of staggering proportions. The statistics speak for themselves:

  • In the early 1900s, one in 20 people developed cancer
  • In the 1940s, one in 16 people developed cancer
  • In the 1970s, it was one in 10
  • Today, it’s one in three!

According to the CDC, about 1,660,290 (1.66 million) new cancer cases are expected to be diagnosed in 20131. If overall death rates are falling, why are incidence rates still on the rise? The answer is simple: the 40-year “war on cancer” has been a farce.

The cancer epidemic is a dream for Big Pharma, and their campaigns to silence cancer cures have been fierce, which is a tale well told in the documentary film featured below, Cancer: Forbidden Cures.

The Cancer Machine

Why Medicine Won’t Allow Cancer to Be CuredPlease understand that cancer is big business. The cancer industry is spending virtually nothing of its multi-billion dollar resources on effective prevention strategies, such as dietary guidelines, exercise and obesity education. Instead, it pours its money into treating cancer, not preventing or curing it.

Why would they shoot their cash cow? If they can keep the well-oiled Cancer Machine running, they will continue to make massive profits on chemotherapy drugs, radiotherapy, diagnostic procedures and surgeries.

The typical cancer patient spends $50,000 fighting the disease. Chemotherapy drugs are among the most expensive of all treatments, many ranging from $3,000 to $7,000 for a one-month supply.

If the cancer industry allows a cure, then their patient base goes away. It makes more sense to keep a steady stream of cancer patients alive, but sick and coming back for more. How did this societal monster come about?

The featured documentary is enormously informative. It details how the pharmaceutical industry partnered with the American Medical Association (AMA) in an ingenious plan to overtake the medical system in four swift, easy steps, back in the early 1900s. In a nutshell, it went something like this:

  1. International bankers that own the drug and chemical companies gained control over the medical education system over 100 years ago.
  2. They gave grants to the AMA and leading medical schools in exchange for seats on their board and the ability to control policy.
  3. Finally, they cleverly engineered their control of virtually every federal regulatory agency relating to the practice of medicine.


‘Don’t You DARE Cure Anyone!’

In spite of the enormous amounts of money funneled into cancer research today, two out of three cancer patients will be dead within five years after receiving all or part of the standard cancer treatment trinity—surgery, radiotherapy and chemotherapy. This is not too surprising when you consider that two of the three are carcinogenic themselves! One study estimated that chemotherapy benefits about one of every 20 people receiving it.

Over the last hundred years, a number of natural cancer treatments have been developed and used successfully to treat patients in the US and other countries. All have been vehemently discounted, silenced, and pushed under the rug by the medical monopoly, with physicians and researchers attacked, smeared, sent to prison, and professionally ruined for daring to defy the medical establishment.

To this day, with respect to credibility in medicine, “quack” is synonymous with “competition.”

In order to protect the medical monopoly, any viable natural treatment is met with massive opposition by the pharmaceutical and medical industries. Drug companies have no interest in natural agents that they cannot patent, because they interfere with their revenue stream. They will go—and have gone—to extreme measures to prevent the truth about effective natural treatments (competitive threats) from reaching the public.

The FDA is now, thanks to PDUFA, primarily funded by the drug companies and is complicit in this process. They restrict competition in the guise of protecting the public, when the reality is they are protecting the profits of the drug companies.

My Top 12 Cancer Prevention Strategies

There is so much you can do to lower your risk for cancer. But please don’t wait until you get the diagnosis—you have to take preventative steps NOW. It’s much easier to prevent cancer than to treat it, once it takes hold. I believe you can virtually eliminate your risk of cancer and chronic disease, and radically improve your chances of recovering from cancer if you currently have it, by following these relatively simple strategies.

  1. Food Preparation: Eat at least one-third of your food raw. Avoid frying or charbroiling; boil, poach or steam your foods instead. Consider adding cancer-fighting whole foods, herbs, spices and supplements to your diet, such as broccoli,curcumin and resveratrol. To learn more about how these anti-angiogenetic foods fight cancer, please see our previous article: “Dramatically Effective New Natural Way to Starve Cancer and Obesity.
  2. Carbohydrates and Sugar: Reduce or eliminate processed foods, sugar/fructose and grain-based foods from your diet. This applies to whole unprocessed organic grains as well, as they tend to rapidly break down and drive up your insulin level. The evidence is quite clear that if you want to avoid cancer, or you currently have cancer, you absolutely MUST avoid all forms of sugar, especially fructose, which feeds cancer cells and promotes their growth. Make sure your total fructose intake is around 25 grams daily, including fruit.
  3. Protein and Fat: Consider reducing your protein levels to one gram per kilogram of lean body weight. It would be unusual for most adults to need more than 100 grams of protein and most likely close to half of that amount. Replace excess protein with high-quality fats, such as organic eggs from pastured hens, high-quality meats, avocados, and coconut oil.
  4. GMOs: Avoid genetically engineered foods as they are typically treated with herbicides such as Roundup (glyphosate), and likely to be carcinogenic. A French research team that has extensively studied Roundup concluded it’s toxic to human cells, and likely carcinogenic to humans. Choose fresh, organic, preferably locally grown foods.
  5. Animal-Based Omega-3 fats: Normalize your ratio of omega-3 to omega-6 fats by taking a high-quality krill oil and reducing your intake of processed vegetable oils.
  6. Natural Probiotics: Optimizing your gut flora will reduce inflammation and strengthen your immune response. Researchers have found a microbe-dependent mechanism through which some cancers mount an inflammatory response that fuels their development and growth. They suggest that inhibiting inflammatory cytokines might slow cancer progression and improve the response to chemotherapy. Adding naturally fermented food to your daily diet is an easy way to prevent cancer or speed recovery. You can always add a high-quality probiotic supplement as well, but naturally fermented foods are the best.
  7. Exercise: Exercise lowers insulin levels, which creates a low sugar environment that discourages the growth and spread of cancer cells. In a three-month study, exercise was found to alter immune cells into a more potent disease-fighting form in cancer survivors who had just completed chemotherapy. Researchers and cancer organizations increasingly recommend making regular exercise a priority in order to reduce your risk of cancer, and help improve cancer outcomes. Research has also found evidence suggesting exercise can help trigger apoptosis (programmed cell death) in cancer cells. Ideally, your exercise program should include balance, strength, flexibility, high intensity interval training (HIIT). For help getting started, refer to my Peak Fitness Program.
  8. Vitamin D: There is scientific evidence you can decrease your risk of cancer by more than half simply by optimizing your vitamin D levels with appropriate sun exposure. Your serum level should hold steady at 50-70 ng/ml, but if you are being treated for cancer, it should be closer to 80-90 ng/ml for optimal benefit. If you take oral vitamin D and have cancer, it would be very prudent to monitor your vitamin D blood levels regularly, as well as supplementing your vitamin K2, as K2 deficiency is actually what produces the symptoms of vitamin D toxicity. To learn more, please see my previous article: “What You Need to Know About Vitamin K2, D and Calcium“.
  9. Sleep: Make sure you are getting enough restorative sleep. Poor sleep can interfere with your melatonin production, which is associated with an increased risk of insulin resistance and weight gain, both of which contribute to cancer’s virility.
  10. Exposure to Toxins: Reduce your exposure to environmental toxins like pesticides, herbicides, household chemical cleaners, synthetic air fresheners and toxic cosmetics.
  11. Exposure to Radiation: Limit your exposure and protect yourself from radiation produced by cell phones, towers, base stations, and Wi-Fi stations, as well as minimizing your exposure from radiation-based medical scans, including dental x-rays, CT scans, and mammograms.
  12. Stress Management: Stress from all causes is a major contributor to disease. Even the CDC states that 85 percent of disease is driven by emotional factors. It is likely that stress and unresolved emotional issues may be more important than the physical ones, so make sure this is addressed. My favorite tool for resolving emotional challenges is Emotional Freedom Techniques (EFT).


What to Do If You Already Have Cancer

Without a doubt the most powerful essential strategy I know of to treat cancer is to starve the cells by depriving them of their food source. Unlike your body cells, which can burn carbs or fat for fuel, cancer cells have lost that metabolic flexibility. Dr. Otto Warburg was actually given a Nobel Prize over 75 years ago for figuring this out but virtually no oncologist actually uses this information.

You can review my recent interview with Dr. D’Agostino below for more details but integrating a ketogenic diet with hyperbaric oxygen therapy which is deadly to cancer cells debilitated by starving them of their fuel source would be the strategy I would recommend to my family if they were diagnosed with cancer.

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