Statin scam exposed: Cholesterol drugs cause rapid aging, brain damage and diabetes – Complete Health and Happiness http://complete-health-and-happiness.com/statin-scam-exposed-cholesterol-drugs-cause-rapid-aging-brain-damage-and-diabetes/?t=MAM
‘Marijuana Tampon’ Might Be the End of Your Period Cramps | Alternative Health http://alternativehealth.press/marijuana-tampon-might-be-the-end-of-your-period-cramps
Asthma affects about 300 million people worldwide. It is growing by 50 percent every decade and causes upwards of 180,000 deaths per year. The cause is not well-understood but here are 8 proven ways to help relieve symptoms naturally.
There is nothing more terrifying than not being able to breathe. But that’s what asthmatics face every day. Asthma is a chronic disease characterized by inflammation of the airways. Symptoms include wheezing, shortness of breath, chest tightness and cough. According to the Global Initiative for Asthma it affects an estimated 300 million people worldwide. And it increases globally by 50 percent every decade.[i]
Asthma is also deadly. According to the World Health Organization, it is linked to more than 180,000 deaths per year.[ii]
No single cause of asthma has been identified. Symptoms may be triggered or worsened by viral infections, allergens, tobacco smoke, exercise and stress, among other things.
Obesity is also linked to asthma. A study from the Harvard School of Public Health found that obesity is both a risk factor for asthma and is associated with increased severity of the symptoms.
And a study in the journal Allergy looked at data from the 2005-2006 National Health and Nutrition Examination Survey (NHANES). It found that obese people had more than 2.5 times the risk of developing asthma as people with a normal body mass index (BMI).
Researchers from Duke University also reviewed NHANES data from 2001 through 2004. They found that people with a BMI in the obese range were 12 percent more likely to have more severe asthma. They hypothesized that the inflammation induced by obesity may contribute to worse asthma symptoms.
Several studies link some asthma cases to childhood vaccines and their timing. In a study of 1,531 children in Manitoba, Canada, researchers found that the risk of developing asthma by the age of seven was cut in half when the first diphtheria, pertussis, tetanus (DPT) immunization was delayed by more than two months. Delaying all three doses of DPT vaccines cut asthma risk by 60 percent.
Studies show breast feeding reduces the risk of developing asthma. In a study of 1500 infants and pre-schoolers, children who were exclusively breast-fed had lower asthma rates than those partially breast-fed or given formula milk.
And a meta-analysis of 12 studies published in the Journal of Pediatrics showed that exclusive breast feeding during the first three months after birth reduced asthma risk by 30 percent. Researchers attributed the effect to the immunomodulatory properties of breast milk.
For those suffering with the disease, there are natural remedies to relieve symptoms. Here are just eight proven ways to relieve asthma.
1. Breathing Exercises
Many studies show that breathing exercises have a therapeutic role in the treatment of asthma. In one randomized controlled trial asthma patients taught breathing exercises showed significant improvements in their quality of life, symptoms, and psychological well-being after six months.
In another study, patients were taught breathing exercises known as the Buteyko Breathing Method named after the Russian physician who developed the technique. Buteyko breathing exercises increased asthma control 40 percent to 79 percent and significantly reduced the use of corticosteroid inhalers compared with a control group.
In addition, yoga breathing exercises are therapeutic for asthma sufferers. In a study of 60 patients, half were randomized to receive yoga breathing instructions. After two months the yoga group showed a statistically significant improvement in lung function as well as improved quality of life.
In another study of 17 university students half the subjects were taught a set of yogic breathing exercises, physical postures, and meditation three times per week. After 16 weeks, data showed that the yoga significantly improved relaxation, led to a more positive attitude, and reduced use of inhalers. The researchers concluded that yoga techniques seem beneficial as an adjunct to the medical management of asthma.
Studies show that one of the active components in the spice turmeric, curcumin, inhibits the allergic response. Other research suggests that curcumin works by preventing or modulating inflammation and oxidative stress in the airways.[iii]
In one study 77 patients with mild to moderate bronchial asthma were randomly assigned to two groups. One group received standard asthma treatment while the other group received the standard therapy plus 500 mg per day of curcumin. After 30 days researchers concluded thatcurcumin significantly helped improve airway obstruction and suggested that curcumin is effective and safe as an add-on therapy for the treatment of bronchial asthma.
Researchers from Brown University School of Medicine tested intravenous magnesium on pediatric patients with moderate to severe asthma. Thirty patients were randomly assigned to receive either 40 mg/kg of magnesium sulfate or a saline solution. Just twenty minutes later themagnesium group showed remarkable improvement in short-term lung function.
Taking magnesium orally is also effective for asthma control. In a study published in theJournal of Asthma 55 patients were randomly assigned to take 340 mg (170 mg twice a day) of magnesium or a placebo. After 6.5 months the magnesium group had better bronchial reactivity, and better subjective measures of asthma control and quality of life.
In another study from Brazil 37 patients all received inhaled fluticasone (brand name Flonase) twice a day and the asthma drug salbutamol as needed. Half the group also took 300 mg per day of magnesium. After two months bronchial reactivity improved significantly in the magnesium group only. The magnesium group also had fewer instances of worsening asthma and used less salbutamol compared to the placebo group.
4. Vitamin D
Asthma has been linked to lower levels of vitamin D. In a study of 483 asthmatics under 15 years of age and 483 matched controls, researchers found that vitamin D deficiency was more prevalent in asthmatics.
A review of vitamin D studies found vitamin D and its deficiency have a number of effects in the body which could affect the development and severity of asthma. Researchers concluded thatvitamin D may improve lung function and response to steroids, and reduce airway remodeling.
And in a double blind, randomized, comparative study, 140 patients received standard treatment for asthma while half also received 1000 mg per day of vitamin D3. After six months researchers found that the vitamin D3 significantly improved the quality of life for severe asthmatics.
Many people find their asthma symptoms disappear on a dairy elimination diet.
A meta-analysis of data from more than 30 studies in the journal Nutrition Reviews found high intake of fruit and vegetables may reduce the risk of asthma and wheezing in adults and children. Researchers concluded that eating more fruit and vegetables could reduce the risk of asthma in adults and children by 46 percent.
Another study found tomatoes particularly powerful. Researchers in Australia had asthmatic adults eat a low antioxidant diet for 10 days. Measures of asthma severity worsened. Then for seven days the patients were randomized to receive either a placebo, tomato extract (45 mg lycopene/day), or tomato juice (45 mg lycopene/day). Patients receiving tomato extract or tomato juice reduced their signs of asthma. The researchers suggested that lycopene-rich supplements should be further investigated as an asthma therapy.
And research from Johns Hopkins University found that sulforaphane, or foods rich in sulforaphane like broccoli, may be adjuvant treatments for asthma. Sulforaphane is an antioxidant and anti-inflammatory phytochemical also found in other cruciferous vegetables like Brussels sprouts, cabbage, cauliflower, bok choy, kale, collards, broccoli sprouts, arugula, and watercress.
6. Fish Oil
Many studies show that fish oil relieves chronic inflammation like that found in asthma. In one study of 20 asthmatic patients, researchers compared fish oil to montelukast (brand name Singulair). Montelukast is a drug used to prevent the wheezing and shortness of breath caused by asthma.
The subjects were randomly assigned to receive either 10 mg of montelukast tablets or 10 fish oil capsules totaling 3.2 g EPA and 2.0 g DHA every day for three weeks. Thereafter all the subjects received both treatments together for another three weeks. Results showed that montelukast and fish oil were equally effective (and fish oil was slightly better) at reducing airway inflammation.
7. Pine Bark
Pycnogenol® is a standardized extract of French maritime pine bark with anti-inflammatory properties. Italian researchers compared it to the use of corticosteroid inhalers for relieving asthma symptoms. A total of 76 patients used an inhaler. Half the group also received 50 mg of Pycnogenol morning and evening.
After six months 55 percent of the Pycnogenol patients were able to reduce their inhaler use compared to only six percent of the control patients. In addition, none of the Pycnogenol patients had a worsening condition but 18.8 percent of the inhaler-only group deteriorated. Researchers concluded that Pycnogenol was effective for better control of allergic asthma and reduced the need for medication.
Also, Pycnogenol is effective to help manage mild-to-moderate childhood asthma. In a randomized, placebo-controlled, double-blind study, 60 subjects, aged 6-18 years old, were given either Pycnogenol or placebo. After three months, the Pyconogenol group had significantly more improvement in lung functions and asthma symptoms. They were also able to reduce or stop their use of rescue inhalers more often than the placebo group.
8. Vitamin B6
In a double-blind study of 76 asthmatic children, patients received 200 mg daily of vitamin B6 (pyridoxine). After five months researchers found vitamin B6 led to significant improvements in asthma symptoms and reduction in dosage of bronchodilators and cortisone.
For more information on the underlying causes, triggers, and reversal of asthma read “Asthma Solved Naturally.”
Recent conversations have centered around the seemingly growing chaos we see around the globe. I would suggest that the chaos is not growing, but rather, our conscious awareness of it, and the possibility that more and more dirt is being hidden under fewer and fewer rugs. In other words, no matter what I/you/we/they have attempted to cover up, from the personal to the planetary, political platform or prison, whether yesterday or a thousand years ago, it’s time for a big housecleaning – for revealing and releasing all the ways that don’t work, don’t fit, aren’t sustainable, that we can’t sneak in our pockets past this particular tollbooth, no matter how many times we shuffle our nickels, how im/patiently we wait in line.
In the midst of all the details, the bigger picture can get lost, and it can be easy to forget that, in a sense, the chaos is relative. That is, relative to how attached we are to how things have been, how we think things should be.
Earlier this afternoon a remembered visual brought home this realization in an almost too-perfect way. Stopped momentarily in my car, I relaxed to the left for a second, resting my head against the doorframe. Immediately my surroundings went blurry as the vibration from the motor rocked my world and everything in it. Raising my head, everything cleared and returned to center. Hmmmm, let me try that again. Yes, there it went…
And in a flash, I remembered a scene from Contact, a sci-fi must-see starring Jodie Foster. It’s been a while, so bear with me if I butcher the story, but in brief, SETI had received a mathematically based signal from far out in space that was eventually decoded into plans for some kind of a machine. No one knew what it was exactly, but the assumption was that it was some type of spaceship that could link us beyond, perhaps to the infinite – or at least the sender of the signal. Not without disagreement, they set to work, and billions of dollars and a few curve balls later, the machine was ready. Jodie was to be the solitary occupant of a spacecraft built for one. Boarding on the big day, Jodie’s character was taken aback to see that a captain’s chair had been added, bolted above and below to the structure, which otherwise was an empty, room-sized sphere. Everyone assured her it had to be that way, it was just for safety – probably an OHSA requirement for some of that funding.
I won’t ruin the story in case you haven’t seen it, but here’s the part that came to me today: When the countdown started and Jodie’s character began her journey, the machine began to vibrate, more and more violently, as speed increased and time began to blur. It looked and sounded like the ship would tear itself apart, rivets flying in every direction, far past the stress limits of the materials and construction.
But then something happened: Jodie’s character dropped the compass she’d brought along, a talisman from her father, a gift of great personal meaning. In the midst of the chaos, she remained present enough to notice that the compass was simply floating in midair – zero gravity had been achieved. Intrepid adventurer that she was, Jodie took a calculated risk, unlatching her shoulder harness – and calmly, easily, lightly floated away from the command chair that had by now begun to violently tear itself from the surrounding structure. Seconds later its bolts came loose, and the whole thing was sucked to the ceiling in a magnetic vacuum of safety. So what was the problem? It wasn’t the machine. It wasn’t Jodie. It wasn’t the compass.
It was the one thing that had been added to the plan: an Attachment. One that kept her rooted in place, in an illusory space of safety.
The second she let go of that attachment – unbuckling the hold it had on her, that she had on it – she was in a space of calm and ease. Ironically, the mad vibrations were destroying only one thing: that safety harness equipped chair.
So here’s my theory, should you care to experiment: If you happen to find yourself in chaos today, or tonight, tomorrow, or even next week (and only if you are not in a moving vehicle): Unbuckle that seatbelt. Whatever it means to you, whatever is holding you in place. Step away from the attachment, whatever it may be – a person, a place, a paradigm, a new vision of you, an old vision of you, how you think things should be, or were, or even better, how someone else thinks things should be – all those things we carry around with us in simple masquerade of our need to control – or at least to think we can. If you’re not sure how to begin, maybe just a willingness to consider the possibility is a good place to start.
Step away. Let go. That attachment was never meant to be in the first place – it wasn’t part of the original plans. Our little ego minds just got smart and thought they’d throw that in there for good measure.
I’ll be doing my best to do the same, although I’m not yet sure just how that will take shape in various situations and settings. But I’ll give it my best shot, one situation at a time, one person at a time, one conversation at a time. Letting go. Letting go. Letting go. Stepping away from all expectations – of how things are, how things were, how things could be, how we could be. Being with what is, in this instant, and now, in this one, and here again, in this next one. Seeing that, first and foremost. And then, to look for that next right step that will always reveal itself.
Hey, if you try this – let me know how it turns out. I’ll see ya on the flip side.
Seven years ago, when I emerged from the doctor’s office after having my worst fears confirmed, I knew that I had some big decisions to make. With the words “most likely Multiple Sclerosis”reverberating around my mind I felt shaky and nauseas. I wanted to crawl into bed and drown in a deluge of tears and despair. This was not how I had envisaged my charmed life unfolding. I was with the love of my life and we had a nine month old baby; this was supposed to be the happily ever after part.
That night lying in the bath I could feel my body throbbing with fear. My mind was busy fielding the images that surfaced and threatened to overtake me: the slow degeneration of my body, the social pity, and all the dreams that would now never be. Then, at one point I felt compelled to raise my arms in the air and slowly wave them around. Though I knew it seemed mad, the urge was strong and I followed the call. As I let a slow organic flow move my upper limbs I felt the first glimpse of hope since I left the doctor’s office. This movement that emanated from deep within seemed to calm my mind and made me feel that maybe this illness had deeper implications.
The next day I decided to face my fears head on. I had been doing extensive research trying to figure out what might be going on with my body. Though references to MS had often appeared in my searches of symptoms, up until then I largely ignored them, thinking that there was no way such a fate could be mine. Now that I knew the reality, I delved in and began exploring the mysterious, frightening world of MS.
One of the first things that struck me was the way most people seemed to speak of MS, and disease in general, as something evil that had to be beaten. This did not fit into my world view. Fighting felt like a low vibration approach that promoted a dualistic view of myself in relation to the illness. I wanted a loving approach, not a fear based one.
Just before my symptoms had manifested I had been doing some deep emotional work and had finally accessed an elusive wounded aspect hiding down in the dark recesses of my being. I had promised this part that I would no longer neglect it; I would work to heal. Though I had seen this as a metaphorical situation, it appeared that my body had taken it more literally; this crippled aspect had risen to the surface and could no longer be ignored.
When I looked at my illness from this perspective, as a physical manifestation of emotional wounds, the idea vilifying it, seeing it as something to dominate and conquer felt backwards and cruel. I decided instead that I would see my condition as a teacher instead of an enemy. I had long perceived my life as a magical journey in which I was connected to and guided by the universe. Initially this situation had made me feel as though I had been forsaken, but the more I allowed myself to accept my circumstances, the more it made sense. This condition hadn’t occurred as a punishment, or because I had done something wrong, but because it had something to teach me, and though it seemed highly undesirable, it actually had many gifts to offer.
Here are some of the things I have learned:
I learned to honour my body. I had long been interested in health, but developing MS symptoms forced me to take this to a new level. I realized how often I neglected the gentle calls of my body when they were inconvenient. Even a simple message like ‘I’m thirsty,’ would often be ignored if my mind was happily engaged in some pursuit or other. Now my body became like a highly sensitive machine that if not cared for would immediately see an exacerbation of my symptoms. Along the way I have learned so much about health and nutrition and the personal requirements of my mind and body that in many respects I actually feel healthier now than I did before MS entered my life.
I learned the effect that fear has on my physical, mental and spiritual well being. When I allow myself to wallow in self-pity and fear, symptoms quickly worsen; if I focus on the positive things in my life that give me joy, I feel better. I have a gorgeous, amazingly supportive partner; a beautiful, healthy child; friends and family who love and care for me; and generally so much to be grateful for. This is where I need to put my energy and focus.
I learned to trust myself at an even deeper level. Though specialists encouraged me to follow the standard medical protocol, the idea of stifling the symptoms, which I had come to see as a means for my body to communicate with me, felt intuitively wrong. How could I honour the needs of my body, if communication was disabled? I knew there was no absolute right or wrong approach. Different people have different paths and the important thing is that we do what feels right for us on an individual basis. Though initially it felt daunting to ignore the authority of the conventional medical profession, I gradually came to trust that my body has a wisdom of its own. I felt empowered and gained confidence in my ability to discern what was right for me. I still take guidance from professionals, but not if it doesn’t feel in alignment with my inner voice.
I learned that I don’t have to be defined by my illness. Having a chronic condition can sometimes feel all consuming, but there is so much more to who I am than my symptoms. Everyone has trials and tribulations in life, and we all have choices in terms of how we approach them. We can either allow our issues to define us, or we can use them as tools to help guide us into alignment with a more loving, more whole, and more meaningful version of ourselves.
Finally, I learned the importance of trusting and embracing the process. Some days I felt great and it was easy to feel optimistic, others days my symptoms would worsen without any clear reason, and I would find myself going into fear. With time I learned to stop worrying, even when things appear not to be going in the direction I desire. Sometimes the healing process requires symptoms to worsen before they get better, other times I just need to pay a bit more attention to what my body is telling me, and either way, allowing myself to become overwhelmed by fear never helps anything. Sometimes things happen for reasons that only become clear later. For example, one year when my symptoms took a turn for the worse I had to spend a lot of time in bed. Unable to do many of the things that previously would have filled my day I began to write. During that year I ended up writing a book — Jump Into The Blue — that I’d always felt called to share. So while it was difficult on many levels, that year was pivotal in pushing me towards manifesting my dreams. Similarly, my husband through researching ways to assist me ended up discovering his own life passion. He has become an expert in the art of fermentation and now teaches others how to use these ancient techniques to promote their own health.
In reality there is no perfect fairytale happily ever after. I believe we are here to learn and grow, and whether we like it or not, it is often life’s challenges that push us to dig deeper and find our own personal gold. I think ultimately the important thing to remember when faced with any trial is that we have a choice in terms of perspective, and the choice we make will define the experience. If we adopt a positive, empowering approach we will be much more likely to uncover the gifts that lie beneath the surface of the hurdles we are faced with
“Life’s challenges are not supposed to paralyze you, they’re supposed to help you discover who you are.” ~ Bernice Johnson Reagon
When it comes down to it, corporations have just one purpose: turn a large profit for shareholders. So it should come as no surprise that corporate America has turned its eye toward the wildly successful recreational cannabis movement. In 2015 alone, legal pot sales in the United States jumped 17% to $5.4 billion and are expected to grow 25% this year — an estimated $6.7 billion in U.S. sales. And, of course, corporate interests want their share.
But it’s not simply a modern day gold rush, many feel there is an underlying agenda at hand — with a few very wealthy individuals pulling the strings.
Exploding cannabis market
The numbers behind legalized pot sales are astonishing considering the first recreational dispensaries opened for business in Colorado a mere two years ago. And this is only the beginning.
According to ArcView Market Research, the legal cannabis market will be worth a staggering $21.8 billion total sales by 2020. To put this in perspective, when the legalized marijuana market hits that point, it will surpass the National Football League, which is estimated to reach $25 billion by 2027.
“I think that we are going to see in 2016 this next wave of investors, the next wave of business operators, and people who’ve sort of been watching or dipping their toe in, really starting to swing for the fences and take it really seriously,” said ArcView CEO Troy Dayton.
Four states and the District of Columbia have legalized recreational marijuana to date, with a total 23 states having legalized medical marijuana. A half dozen additional states are considering opening up legalized recreational use with ballot measures in autumn 2016. California is one state that is being watched with great anticipation, where a large portion of the total industry is accounted for with medical marijuana sales. If the state legalizes recreational marijuana this November, the sky is the limit for industry sales. Add to this the revamping of recreational cannabis legislation in Colorado, Oregon and Washington — which will allow investors from around the U.S. to finance the legal marijuana industry — and it’s no wonder corporate heavy hitters are attentive.
But it’s not all glory for the movement, there are those who feel there’s a hidden agenda, one that is controlled by corporate interests and a few exceptionally wealthy individuals who have ties to the biotech industry and Big Pharma.
Hidden motives for legalized cannabis
Portrayed as a grassroots movement, you may not realize that the driving force behind legalization of both medical and recreational marijuana is none other than billionaire George Soros. Along with a cadre of other wealthy donors, Soros has spent at least $80 million in the quest of legalizing cannabis. Through his Foundation to Promote an Open Society, he has funneled donations to the Drug Policy Alliance — a non-profit organization with “the principal goal of ending the American “War on Drugs.”’ He’s also donates annually to the American Civil Liberties Union, an organization which funds marijuana legalization efforts, and gives to the Marijuana Policy Project, which supports state ballot measures.
In the official paper, “DEA Position on Marijuana” the agency points out that “a few wealthy businessmen — not broad grassroots support — started and sustain the ‘medical’ marijuana and drug legalization movements in the U.S. Without their money and influence, the drug legalization movement would shrivel.”
Moreover, Soros was instrumental in Uruguay becoming the first country to legalize cultivation, sale and consumption of marijuana. He sits on the board of the Drug Policy Alliance (DPA) — the world’s most powerful organization regarding the legalization of marijuana. The DPA is not only extremely influential in the United States, but also in Uruguay and other Latin American countries.
George Soros appears to be a hero of the cannabis movement — helping states (and even an entire country) pass legislation for medical and recreational use. But he also has strong ties with the pharmaceutical industry and Monsanto as a major stock holder — the same chemical corporation and biotech giant that developed Agent Orange, DDT, PCBs, toxic pesticides, rBGH, Roundup Ready and genetically engineered Frankenfoods. While the widely circulated urban myth that Monsanto is currently developing genetically modified cannabis is false, if you dig a bit deeper, it turns out the corporation quietly conducts research projects on tetrahydrocannabinol (THC) found in marijuana for the apparent purpose of genetically modifying the plant at a future date.
Writes William Engdahl, author of Seeds of Destruction: The Hidden Agenda of Genetic Manipulation:
“David Watson of the Dutch company Hortapharm has since 1990 created the world’s largest collection of Cannabis seed varieties. In 1998, the British firm GW Pharmaceuticals signed an agreement with Hortapharm that gives GW Pharma the rights to use the Hortapharm cannabis for their research.
In 2003 the German Bayer AG then signed an agreement with GW Pharmaceuticals for joint research on a cannabis-based extract. In 2007, Bayer AG agreed to an exchange of technology with… Monsanto. […] Thus Monsanto has discreet access to the work of the cannabis plant and its genetic modification. In 2009 GW Pharmaceuticals announced that it had succeeded in genetically altering a cannabis plant and patented a new breed of cannabis.”
Engdahl believes it doesn’t take a large stretch of the imagination to see that Monsanto could very well be laying the groundwork for a future controlling patented cannabis seeds, especially since Uruguay’s president Mujica has expressed a desire to create unique genetic codes for marijuana within his country in order to undermine the blackmarket. Monsanto could easily step in and accommodate Mujica’s wish, considering the history the biotech corporation has had in Uruguay over the decades growing GMO soybeans and maize.
Back in the United States, it’s interesting to note that the criminalization of both industrial hemp and marijuana has been financially lucrative for a range of industries, like the prison system and military-industrial complex, along with the banking, fossil fuel, timber, cotton and pharmaceutical industries. In light of this, Conrad Justice Kiczenski warns:
“The next stage in continuing this control is in the regulation, licensing and taxation of Cannabis cultivation and use through the only practical means available to the corporate system, which is through genetic engineering and patenting of the Cannabis genome.”
We often hear that correlation is not causation, so why are world renowned scientists treating the Zika virus as the sole cause of microcephaly in Brazil when there are so many other factors?
Since the virus Zika was blamed for a cluster of cases in northeastern Brazil of the devastating birth defect microcephaly, the mainstream media have been dominated by fear of a Zika pandemic. Meanwhile, the real culprit(s) behind the surge in microcephaly in that corner of Brazil have been ignored, with the exception of a few scientists, and even fewer journalists.
This story started on Feb. 1, 2016, when the World Health Organization (WHO) announced a pandemic emergency with the Zika virus, a much milder cousin of Dengue fever. The WHO blamed Zika alone for the sharp uptick in microcephaly (shrunken heads, resulting in shrunken and undeveloped brains, with a wide range of symptoms and disabilities possible, depending on the severity of the case) in babies born in impoverished areas of northeast Brazil.
Politics and an unscientific approach ensued, with the Centers for Disease Control (CDC) and National Institutes of Health (NIH) joining forces with the WHO. The twin U.S. health care agencies launched a propaganda campaign of fear to justify a money grab from U.S. taxpayers. But for a change, Congress developed a backbone and denied the pleas of President Obama, CDC Director Tom Frieden, and NIHDirector Anthony Fauci, who were seeking $1.9 billion for Zika vaccine R&D.
On June 28, the “Zika Bill” was blocked by Senate Democrats, due to issues over the “provisions of the bill,” but apparently not the lower price tag of $1.1 billion.
Missing CSI Investigation
Whether it’s a failed structure or a broken marriage, it often isn’t one item alone that causes the collapse, but a series of them in a cascade of negative events that does the final damage.
Instead of announcing the Zika pandemic, the three international health agencies should have launched a CSI-type investigation in that quarter of Brazil examining all of the environmental triggers and toxinsthat might be contributing to the surge in microcephaly. But that didn’t happen.
“The increase in microcephaly in that part of the world is unique to Brazil. You don’t see rate increases anywhere else,” Dr. James Lyons-Weiler said in a telephone interview on the likely suspects causing the rise in deformed fetuses and babies.
He explained that the “interactions between two or more of the potential causal factors are rarely ever studied by CDC’s scientists. They are not very good with studying interactions,” which might be the underlying cause of an infectious disease or spread of a virus.
Author and research scientist Lyons-Weiler’s early problem solving skills in recognizing the utility in information in DNA-hybridization led him into deeper research on the evolution of diseases, cancer, and mammals.
In 2015, Dr. Lyons-Weiler launched the Institute for Pure and Applied Knowledge (IPAK), a non-profit organization that since its inception has been challenging half-baked science taken as gospel.
In a co-authored paper that Lyons-Weiler led, his scientific team identified nine likely suspects for the rise in microcephaly. The unpublished paper to date, “Areas of Research and Preliminary Evidence onMicrocephaly, Guillain-Barré Syndrome and Zika Virus Infection in the Western Hemisphere,” outlined the suspects.
They range from “Direct Zika-related microcephaly through unspecified mechanisms” and “molecular mimicry” in two types of vaccines given to pregnant women, to “Glyphosate toxicity in bovine products” leaching into those vaccines, and the unintended outcome of genetically modified (GM) mosquitoes, whose world pilot program was launched in 2012 in that same northeast corner of Brazil by the British concern Oxitec.
“The Zika virus has a protein that matches a human protein within 96 percent. Zika also has an element in its genomic sequence similar to one in other flaviviruses, too, like West Nile to Dengue fever. That means Zika could enter the placenta and blood brain barrier of infants. Yet since there is no increase in acute microcephaly outside of Brazil, if it’s Zika, there may be a missing molecular or chemical co-factor,” Lyons-Weiler explained.
The ability of viruses to produce specific disease symptoms is often known to be modified by co-factors. “Something is different in Brazil,” said Lyons-Weiler.
On June 1, 2015, Denmark, a farming country, banned the sale and use of Monsanto’s ubiquitous weed killer Roundup, as a result of the Danish Environment Authority declaring glyphosate as a carcinogen. Earlier that year, the WHO classified glyphosate as “probably carcinogenic to humans.”
The ban and the statement had little effect on removing the sale of glyphosate-containing productsin the United States and South America. And that has bugged MIT Senior Research Scientist Stephanie Seneff, Ph.D., who conducts research at the MIT Computer Science and Artificial Intelligence Laboratory.
At this year’s Autism One Conference in Chicago, Dr. Seneff presented a 66-slide deck, “Glyphosate, Folic Acid, Neural Tube Defects and Autism,” highlighting potential associations between chemicals, biology, and children susceptible to autism. In mid-June, Seneff presented at a U.S. Congressional hearing on glyphosate, in Washington, D.C.
In an email, Stephanie Seneff wrote: “It is ridiculous that the only thing the research community seems to be focused on with respect to the microcephaly epidemic in NE Brazil is the Zika virus. While the virus may be a factor in the epidemic, there are many other potential factors that deserve at least equal attention. These include:
(1) “Simultaneous exposure to two herbicides—glufosinate and glyphosate—due to the recent introduction of GMO glufosinate-resistant soybeans on top of the glyphosate-resistant soybeans (glufosinate substitution for glutamine during protein synthesis is a direct path to microcephaly via disruption of asparagine synthase);
(2) “The addition of larvicides directly to the drinking water;
(3) “The introduction of the GM mosquitoes from larvae that were likely fed glyphosate-contaminatedsugar and glyphosate-contaminated blood following maturation;
(4) “The heavy use of ethanol as a fuel in the trucks driving through the region (derived from GM Roundup-ready sugar beets or sugar cane sprayed with Roundup just before harvest), and;
(5) “The recent implementation of policies that encourage vaccination of pregnant women with Tdap, flu vaccine, and possibly MMR vaccine. All of these potential contributors should be thoroughly investigated before concluding that Zika is the entire story with the epidemic.”
What do all of these potential triggers mean? Even if they are not the direct cause of microcephaly, they are contributing to both polluting the land and thus plant, animal, and human life. That should give governments around the world pause.
To date, it has not worked out that way yet.
Where there is big opportunity for billions of dollars in profits, there is Big Industry—Big Pharma, Big Agriculture, Big you name it—led by multinational corporations that seek home run-like profits. There are also big governments that either look the other way or are fine with raking in some of those profits, too.
“The timing is wrong for Zika” said Lyons-Weiler, who pointed to a study showing an increase inmicrocephaly in Brazil two years before Zika made it to Brazil.
“What is clear is the experimentation with whole-cell pertussis vaccination in the slums is ongoing, because the population cannot afford the fee for the clinic, where the safer acellular vaccine is available. The increase in microcephaly began one year after Brazil adopted a mandatory prenatal care program, which includes vaccinations during pregnancy,” he concluded.
Zika is not about science. It’s about money and profit at the expense of the people, domestic and foreign.
Importance Acute coronary syndrome (ACS), the acute manifestation of ischemic heart disease, remains a major cause of morbidity and mortality worldwide and is responsible for more than 1 million hospital admissions in the United States annually. Considerable research is being conducted in the field. This review provides a contemporary overview of key new findings on the pathophysiology, diagnosis, treatment, and prognosis of ACS.
Observations While plaque rupture is the most frequent cause of coronary thrombosis, studies with optical coherence tomography demonstrate that superficial plaque erosion is more common than previously thought. High-sensitivity troponin assays (not yet available in the United States) and cardiac computed tomographic angiography are being increasingly used in diagnosis and risk stratification of patients with suspected ACS. New data from long-term dual antiplatelet therapy studies and investigations of anticoagulants provide important insights into the balance between ischemic and bleeding risks. The added benefit of percutaneous coronary intervention in non–infarct-related arteries in patients with ST-segment elevation myocardial infarction has been demonstrated in randomized trials, and the radial approach has become the standard of care in patients with ACS undergoing angiography. Promising old and new adjunctive therapies, such as pretreatment with β-blockers, ezetimibe, and proprotein convertase subtilisin kexin type 9 (PCSK9) inhibitors, are discussed. New guidelines on the management of non–ST-segment elevation ACS were published in the last 2 years, as well as scientific documents on ACS in understudied populations, such as women and patients with renal dysfunction.
Conclusions and Relevance Substantial progress in the prevention, diagnosis, and management of patients with ACS has been accomplished in recent years. Despite optimal pharmacological and invasive therapies, the burden of recurrent ischemic events and mortality remains high, and future research is ongoing to prevent and improve the outcome of patients with ACS.
Originally titled, “A Critique Of Scientific Literature: Pesticides and Polio,” this article by Jim West was first published in The Townsend Letter for Doctors and Patients, June 2000, then republished as a 2nd edition in 2002 by The Weston A. Price Foundation, with additional material and the editorship of Sally Fallon. The article summarizes his book, “DDT/Polio”, which he had attempted to publish in 1998. This is a 3rd edition, August 14, 2015.
It has been alleged that DDT causes or contributes to a wide variety of diseases of humans and animals not previously recognized as associated with any chemical. Such diseases included… poliomyelitis, …such irresponsible claims could produce great harm and, if taken seriously, even interfere with scientific search for true causes… (Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, 1991)
Hayes and Laws were informing their readers about the heretic, Dr. Morton S. Biskind.
In 1953, when Biskind’s writings were published, the United States had just endured its greatest polio epidemic. The entire public was steeped in dramatic images — a predatory poliovirus, nearly a million dead and paralyzed children, iron lungs, struggling doctors and dedicated nurses. The late president Franklin D. Roosevelt had been memorialized as a polio victim who was infected with the deadly poliovirus near the beautiful and remote island of Campobello. The media was saturated with positive images of scientific progress and the marvels of DDT to kill disease-carrying mosquitoes. Jonas Salk was in the wings, preparing to be moved center stage.
Through this intellectually paralyzing atmosphere, Dr. Biskind had the composure to argue what he thought was the most obvious explanation for the polio epidemic: Central nervous system diseases such as polio are actually the physiological and symptomatic manifestations of the ongoing government and industry sponsored inundation of the world’s populace with central nervous system poisons.
Today, few remember this poignant writer who struggled with the issues of pesticides, issues that Rachel Carson would be allowed to politely bring to public awareness nine years later, as the lead story in The New Yorker magazine and then as a national best seller, by limiting her focus to the environment and wildlife. Biskind had the audacity to write about human damage.
I found “M.S. Biskind” in the endnotes to Hayes’ and Laws’ diatribe. What could possibly have motivated Hayes’ and Laws’ biased genuflection towards germ theory? Such offerings, commonly written into the final paragraphs of scientific articles, are usually done with an appearance of impartiality. With great anticipation, I went to a medical library and found Biskind’s 10-page 1953 article in the American Journal of Digestive Diseases. Presented below are excerpts regarding polio from his article.
In 1945, against the advice of investigators who had studied the pharmacology of the compound and found it dangerous for all forms of life, DDT (chlorophenoethane, dichlorodiphenyl-trichloroethane) was released in the United States and other countries for general use by the public as an insecticide.
Since the last war there have been a number of curious changes in the incidence of certain ailments and the development of new syndromes never before observed. A most significant feature of this situation is that both man and all his domestic animals have simultaneously been affected.
In man, the incidence of poliomyelitis has risen sharply;
It was even known by 1945 that DDT is stored in the body fat of mammals and appears in the milk. With this foreknowledge the series of catastrophic events that followed the most intensive campaign of mass poisoning in known human history, should not have surprised the experts. Yet, far from admitting a causal relationship so obvious that in any other field of biology it would be instantly accepted, virtually the entire apparatus of communication, lay and scientific alike, has been devoted to denying, concealing, suppressing, distorting and attempts to convert into its opposite, the overwhelming evidence. Libel, slander and economic boycott have not been overlooked in this campaign.
Early in 1949, as a result of studies during the previous year, the author published reports implicating DDT preparations in the syndrome widely attributed to a “virus-X” in man, in “X-disease” in cattle and in often fatal syndromes in dogs and cats. The relationship was promptly denied by government officials, who provided no evidence to contest the author’s observations but relied solely on the prestige of government authority and sheer numbers of experts to bolster their position.
[“X-disease”] …studied by the author following known exposure to DDT and related compounds and over and over again in the same patients, each time following known exposure. We have described the syndrome as follows:
…In acute exacerbations, mild clonic convulsions involving mainly the legs, have been observed. Several young children exposed to DDT developed a limp lasting from 2 or 3 days to a week or more.
Simultaneously with the occurrence of this disorder [X-disease] a number of related changes occurred in the incidence of known diseases. The most striking of these is poliomyelitis. In the United States the incidence of polio had been increasing prior to 1945 at a fairly constant rate, but its epidemiologic characteristics remained unchanged. Beginning in 1946 the rate of increase more than doubled. Since then remarkable changes in the character of the disease have been noted. Contrary to all past experience, the disease has remained epidemic year after year.
DDT vs Polio (1945-1953)
In the graph below, I provide confirmation of Biskind’s observations for 1945-1953, in terms of polio incidence and pesticide production. I have utilized pesticide data from Hayes and Laws which they had derived from US Tariff Commission data. Polio incidence data was gathered from US Vital Statistics., Although I argue herein against Hayes’ characterization of Biskind’s work, credit goes to Hayes for publishing arcane pesticide data. All graphs refer to paralytic polio.
Biskind also describes physiological evidence of DDT poisoning that resembles polio physiology:
Particularly relevant to recent aspects of this problem are neglected studies by Lillie and his collaborators of the National Institutes of Health, published in 1944 and 1947 respectively, which showed that DDT may produce degeneration of the anterior horn cells of the spinal cord in animals. These changes do not occur regularly in exposed animals any more than they do in human beings, but they do appear often enough to be significant.
He continues, bearing his exasperation in trying to make the obvious plain.
When the population is exposed to a chemical agent known to produce in animals lesions in the spinal cord resembling those in human polio, and thereafter the latter disease increases sharply in incidence and maintains its epidemic character year after year, is it unreasonable to suspect an etiologic relationship?
Before finding Biskind’s work, I had spent months engaged in a nearly futile search for the physiology of acute DDT poisoning. I began to sense that American DDT literature as a whole intends to convey that DDT is not dangerous except with regard to its general environmental effects due to persistent bioaccumulation, and that the physiology of acute DDT poisoning is therefore trivial. DDT literature uniformly jumps from descriptions of symptoms, over physiology, to the biochemistry of DDT-caused dysfunction in nerve tissue.
It was as though detectives had come upon a mass-murder scene and immediately became obsessed with the biochemistry of dying cells around bullet holes, while ignoring the bullet holes.
Eventually, I did find a German study of the physiology of acute DDT poisoning, by Daniel Dresden. (Physiological Investigations Into The Action Of DDT, G.W. Van Der Wiel & Co., 1949) This study confirms that DDT poisoning often causes polio-like physiology:
Conspicuous histological degeneration was, however, often found in the central nervous system. The most striking ones were found in the cerebellum, mainly in the nucleus dentatus and the cortex cells. Among other things an increase of the neuroglia and a necrotic degeneration and resorption of ganglionic cells was found. The Purkinje cells were less seriously affected than the other neurons. Also in the spinal cord abnormalities of a degenerative nature were found.
…such changes were not found invariably… there is neither an obvious relation between the size and spreading of the lesion and the quantity of DDT applied… information of adequate precision about the nature of the anomalies is lacking.
So we find that especially the cerebellum and the spinal cord are histologically affected by DDT.
And more recently, in the works of Ralph Scobey, MD, I found that from ancient times to the early 20th century, the symptoms and physiology of paralytic poliomyelitis were often described as the results of poisoning. It wasn’t until the mid-19th century that the word “poliomyelitis” became the designation for the paralytic effects of both severe poisoning and polio-like diseases assumed to be germ-caused.
In contemporary Britain, a farmer-turned-scientist, Mark Purdey, has found substantial evidence that Mad Cow Disease, a form of polio-like encephalitis, was caused by a government-mandated cattle treatment consisting of organophosphate pesticide and a compound similar to thalidomide. Unlike most scientists, Mark Purdey became legally embroiled with the government during his research.
Morton S. Biskind had the courage to write about humans. His views fell into disfavor after the introduction of the polio vaccines, which was a grand act that proved in most people’s minds that polio was caused by a virus. By October, 1955, Biskind, whose works had been published in established medical journals and who testified before the Senate on the dangers of pesticides, was forced to self-publish his writings, one of which I found while browsing through an old card catalog. A scan of Medline/Pubmed found no other works by him except for a very tame article in 1972, warning that diseases incurred during a patient’s stay in a hospital are not necessarily due to microbes. He died not long thereafter, in his late 60s. I don’t have the precise date of death, though his birth was in 1906.
A Contemporary Study
Below are three graphs that confirm Biskind, utilizing data that spans far beyond his observations. Due to the paucity of data regarding pesticide exposure and locale, these findings of production data are presented as an indication of exposure, keeping in mind the great changes in public awareness and legislation beginning circa 1950, which also served to reduce DDT exposure. Pesticide production data comes from Hayes and Laws.
DDT vs Polio (1940-1970)
In this graph I did not include DDT data for the period of 1954 onward because DDT distribution was then being shifted out of the U.S. and into developing nations, while its U.S. production skyrocketed.
Governmental hearings, including those with Biskind, Scobey and others, brought about greater awareness of DDT dangers, as well as better labeling and handling methods. Due to public governmental debate in 1949-51 and numerous policy and legislative changes afterward, DDT production figures after these dates do not correlate with US usage or exposure to DDT.,,
DDT Before 1950
Before 1950, DDT was hailed as a miracle of progress that was virtually non-toxic to humans, in spite of FDA’s warnings and attempts to keep it off the market. This photo on the left is one of several similar photos from Zimmerman, et al, DDT: Killer of Killers (1946). The advertisement on the right is from an unknown source, though it appears to be circa 1954.
Other photos in Zimmerman advocate 5% DDT solution sprayed directly on dairy cows (body, feed, and water):
This promotion of highly questionable products is reflected in present-day genetically engineered food campaigns.
DDT after 1950
Governmental hearings, including Biskind and Scobey, and others, eventually brought about greater awareness of the dangers, better labeling and handling methods.
DDT after 1954
This period is given special consideration for DDT.
After 1954, DDT production increased tremendously, but mainly as an export product. Due to public governmental debate in 1950-51 and numerous policy and legislative changes afterward, its production figures thereon do not at all correlate with U.S. usage or exposure to DDT.
As many studies demonstrate, DDT exposure after 1954 declined sharply, and this decline is represented in the following graph, along with supporting data. DDT production is not shown, post-1954.
Historical context: DDT was incriminated from 1950 until its registration cancelation in 1968 and ban in 1972. Thus, 1950-1951 represents a point of increased public awareness, changes in legislation and policy, voluntary phase-out, and labeling requirements. It is significant for this comparison of DDT against infantile paralysis, that before the period of increased awareness, DDT was mandated on dairies, yet afterward, ruled out of dairies. Much of the domestic usage was shifted to forestry applications, placing less DDT directly into the food chain.
The visual impact of all the persistent pesticide graphs rests upon the assumption that production correlated with human exposure. Given the lack of regulation and the extreme media hype surrounding DDT before 1953, this is not an unrealistic assumption.
It is clear that post-1954 DDT production did not correlate with human exposure. Yet, it is possible to estimate relative values for exposure post-1954. This can be accomplished by reviewing DDT levels in adipose tissue (National Adipose Tissue Survey, and other studies), considering DDT in imported food, and considering the daily amounts of ingested DDT.
The early trend of National Adipose Tissue Survey’s can be interpolated back to 1944, six years from 1950, the first Survey year, because it is safe to assume that DDT tissue levels were zero in 1944, since DDT was introduced for domestic usage in 1945. The estimate of DDT exposure is a reasonable because DDT has a half-life of about one year. To achieve any downward trend in the DDT/adipose line, DDT exposure had to have decreased sharply.
Note that no scale is provided for “relative DDT exposure”. The Survey values are presented without distortion, linearly, with the starting point at 1954, and values for are estimates based on the Survey and DDT ingestion data.
Error is limited by two boundaries, for the estimated values of DDT exposure. 1) Exposure’s downward slope must be much greater than the Survey line’s downward slope, because of DDT’s half-life. 2) Exposure values must continue at least through 1968.
Hayes and Laws also used a secondary evaluation, DDT intake per day, to explain that from 1954 to 1964-67, DDT ingestion decreased by an approximate factor of five. Significantly, the Salk vaccine program began in 1954.
The observed decrease in the concentration of DDT in food (Walker et al., 1954; Durham et al., 1965a; Duggan, 1968) offers an adequate reason for the decrease in storage in people. The average intake of p,p’-DDT and of total DDT-derived material was 0.178 and 0.280 mg/human/day, respectively, in 1954, but only 0.028 and 0.063 mg/human/day, respectively, during the period 1964-1967. (Hayes and Laws, page 303)
BHC vs Polio (1940-1970)
BHC (benzene hexachloride), a persistent, organochlorine pesticide, is several times more lethal than DDT, in terms of LD50, i.e., the lethal dosage required to kill 50 percent of a test population.
“Unlike the situation with DDT, in which there have been few recorded fatalities, there have been a number of fatalities following poisoning by the cyclodiene and hexachlorocyclohexane-type insecticides. The chlorinated cyclodiene insecticides are among the most toxic and environmentally persistent pesticides known.” (Hayes & Laws)
As shown in the graph below, BHC was produced in 1945-1954 at quantities similar to DDT. In spite of BHC’s lethal quality, it has received much less publicity than DDT. While DDT was banned for such things as an association with the thinning of eagles’ eggs, BHC was phased out of production because it was found, after 15 years, to impart a bad taste to food. It is still used in developing nations. It is tempting to ask whether the highly public DDT was “fronting” for the more dangerous BHC. BHC’s correlation with polio incidence is astonishing.
Lead-Arsenic vs. Polio (1940-1970)
After viewing the DDT and BHC graphs above, note that the period of 1940-46 is unaccounted for in terms of polio-pesticide correlation. The missing piece of the puzzle for this six-year period is supplied by the lead and arsenic compounds. These types of central nervous system (“CNS”) poisons have been the central component of pesticides since their widespread use beginning approximately 1868 until the advent of the organochlorine pesticides in the early 1940s. For those who have thought that “organic” food was the norm before the release of DDT to the civilian sector in 1945, the immense production of lead-arsenic compounds presented in this graph is disappointing. This data requires a reconsideration of any perception regarding “natural” quantities of arsenic found in apple seeds, apricots, or almonds, where pesticides can accumulate systemically from contaminated earth.
Pesticide Composite: Summary
Just over three billion pounds of persistent pesticides are represented in the graph below.
Virtually all peaks and valleys correlate with a direct one-to-one relationship with each pesticide as it enters and leaves the US market. Generally, pesticide production precedes polio incidence by 1 to 2 years. I assume that this variation is due to variations in reporting methods and the time it takes to move pesticides from factory to warehouse, through distribution channels, onto the food crops and to the dinner table.
A composite of the three previous graphs, of the persistent pesticides — lead, arsenic, and the dominant organochlorines (DDT and BHC) — is represented in the following:
These four chemicals were not selected arbitrarily. These are representative of the major pesticides in use during the last major polio epidemic. They persist in the environment as neurotoxins that cause polio-like symptoms, polio-like physiology, and were dumped onto and into human food at dosage levels far above that approved by the FDA. They directly correlate with the incidence of various neurological diseases called “polio” before 1965. They were utilized, according to Biskind, in the “most intensive campaign of mass poisoning in known human history.”
A clear, direct, one-to-one relation between pesticides and paralytic polio over a period of 30 years with pesticides preceding polio incidence in the context of the CNS related physiology just described, leaves little room for complicated virus arguments, even as a co-factor, unless there exists a rigorous proof for virus causation. Polio shows no movement independent from pesticide movement as one would expect for the virus model.
Medical propagandists promote images of a predatory, infectious virus, invading the body and quickly replicating to a level that causes disease, however, in the laboratory, poliovirus does not easily behave in such a predatory manner. Attempts to demonstrate virus causation are performed under extremely artificial and aberrant conditions.
Poliovirus causation was first established in the mainstream mind by publications of an experiment by Landsteiner and Popper in Germany, 1908-1909. Their method was to inject a purée of diseased tissue into two monkeys, “injected into the abdominal cavity”. One monkey died after six days and the other was sickened.
Proof of poliovirus causation was headlined by orthodoxy. This, however, was an assumption — not a proof — of virus causation. The weakness of this method is obvious to everyone except certain viropathologists and has recently been criticized by the molecular biologist Peter Duesberg regarding a modern-day attempt to establish virus causation for kuru, another CNS disease.
Since 1908, the basic test, as intracranial injection, has been repeated successfully many times, using monkeys, dogs and genetically altered mice. However, a crucial weakness exists — polio epidemics do not occur via injections of poliovirus isolate into the brains of the victims through a hole drilled in their skull — except, of course, in laboratories and hospitals.
If injection into the brain is really a valid test for causation then it should serve especially well as a proof for pesticide causation. I propose that pesticides be injected directly into the brains of test animals. If paralysis and nerve degeneration subsequently occur, we then would have proved that pesticides cause polio.
Going further, towards much higher standards of proof than those used to prove virus causation, pesticides could be fed to animals and found to cause CNS disease. This has already been done with DDT and the histology of the spine and brain was poliomyelitis. Virus proofs require injection, often intracranial, to get any reaction from the experimental animal. It is axiomatic that a theory is only as good as its ability to predict future events. I predict that such a test would prove pesticides to be the most reliable causative factor.
The injection of purée of diseased brain tissue into the brains of dogs was the method preferred by Louis Pasteur to establish virus causation with rabies, another CNS disease. A recent, definitive biography of Pasteur finds him to be a most important publicist for germ theory, a crucial promoter for the notion that rabies is caused by a virus. Unfortunately, his rabies experiments were biased and unsupported by independent studies. (G. L. Geison, The Private Science of Louis Pasteur, 1995)
Therefore, in my opinion, even a cofactor theory, where pesticides catalyze predatory poliovirus activity, or where pesticides weaken the immune system to allow opportunistic predatory poliovirus activity, cannot stand up to simple, common sense explanations that include the concept of a symbiotic virus. Neurotoxins are enough of a cause for neurological disease.
The most obvious theory — pesticide causation — should be the dominant theory. But the opposite exists, a pervasive silence regarding pesticide causation juxtaposed against a steady stream of drama regarding virus causation. In light of the evidence presented herein, the silence could ultimately discredit mainstream medical science, institutions of the environmental movement, and the World Health Organization (which directs both DDT application for mosquito campaigns and polio vaccination, world-wide).
When the symptoms of polio are recognized, there is often a claim of virus presence in the body of the polio victim. Sometimes a virus is found. Sometimes that virus is an enterovirus (a virus of the digestive tract). Sometimes that enterovirus is a poliovirus. During polio epidemics, orthodoxy blames the poliovirus, and therefore, my argument for the innocence of the poliovirus requires an explanation of these claims of virus presence and the presence of an agent called the poliovirus. Here are three points:
a) Economic Impetus: During the great epidemic of 1942-1962 polio victims were diagnosed with poliovirus-caused polio, regardless of whether or not the poliovirus was found, because the NFIP (March of Dimes) funds paid only for this kind of polio. Therefore, if patients were going to spend time hospitalized, in iron lungs and undergoing therapy, it would have been economically imperative for the hospital to diagnose them in this way.Thus, presence of poliovirus in poliomyelitis was rarely determined in order to arrive at a diagnosis of polio.
b) Other Pathogens: Even if one believes in virus culpability, other viruses are also claimed by orthodoxy to be the cause of polio-like CNS diseases which are “clinically indistinguishable” from polio. In the 1940-50s, relatively few polio victims were confirmed technically for presence of the poliovirus. In 1958, a laboratory analyses of 222 diagnosed polio victims (Detroit epidemic) found poliovirus in only 51% of the cases. When multiple pathogens are hunted, a mix of pathogens, multiple viruses, fungi, and bacteria, can be associated with a single diagnosed case of polio.
During a polio epidemic, such cases would have likely been diagnosed as “polio”. After the 1970s, with the supposed approaching extinction of the poliovirus, such cases would have been diagnosed as encephalitis or meningitis.
c) Benign Virus: The poliovirus is considered to have been endemic throughout the world going back to ancient times, yet this is not the case with paralytic polio. According to Arno Karlen, author of Man and Microbes, the
“polio virus lives only in people; it probably adapted to the human small intestine countless millennia ago.” He continues, “. . . some historians have claimed that [paralytic] polio goes back to ancient Egypt; it may, but the evidence is thin.”
Karlen makes a lot of sense here in view of the pesticide graphs, Biskind’s arguments, and ancient historians describing paralysis from the inhalation of vaporized chemicals during blacksmithing operations. However, Karlen goes on to write that “the first undisputed case dates from the late eighteenth century.” This statement, however, must be invalid (in its attempt to establish polio images that have a basis in early history) because of Menkes’ statement (above) that other viruses can also be causative for polio symptoms and because common industrial poisons such as arsenic and lead compounds can cause polio-like symptoms. Poisoning by arsenic, as a method of assassination, has also been frequently employed from the earliest eras, and it is not unreasonable to assume that unsuccessful poisonings would have left their victims paralyzed.
Orthodox medical literature can offer no evidence that the poliovirus was anything else than benign until the first polio epidemic, which occurred in Sweden in 1887. This small epidemic occurred 13 years after the invention of DDT in Germany, in 1874, and 14 years after the invention of the first mechanical pesticide crop sprayer, which was used to spray formulations of water, kerosene, soap and arsenic. The epidemic also occurred immediately following an unprecedented flurry of pesticide innovations. This is not to say that DDT was the actual cause of the first polio epidemic, as arsenic was then in widespread use, other organochlorines had been developed, and DDT is said to have been merely an academic exercise.
Poliovirus is categorized as an enterovirus. There are at least 72 known enteroviruses discovered to date. According to Duesberg, many enteroviruses are harmless “passenger viruses.” In view of the material presented here, probably unknown to Duesberg, it is reasonable that we also view poliovirus as harmless outside of extreme laboratory conditions.
The Symbiotic Poliovirus
Having now established the possibility of an innocent poliovirus, its presence in polio can be explained as follows, with five more points:
a) Accelerated Genetic Recombination: Genetic recombination is accelerated whenever a biological system is threatened. Pesticides can be that threat. The proliferation of viruses is known to be part of the process of accelerated genetic recombination.
b) The SOS Response: When a cell is critically threatened, accelerated genetic recombination (which may include virus proliferation) is just one of a set of events that may occur. This set of events is called the “SOS response,” which is known to be triggered by exposure to toxic chemicals or radiation.
Arnold Levine, writing in Field’s Virology, provides an example:
“When lysogenic bacteria were lysed [split open] from without, no virus was detected. But from time to time a bacterium spontaneously lysed and produced many viruses. The influence of ultraviolet light in inducing the release of these viruses was a key observation that began to outline this curious relation between a virus and its host.”
Is this mere irony? Common medical procedures such as chemotherapy, radiation therapy, and the use of toxic pharmaceuticals accelerate genetic recombination and thus the potential for a necessary virus proliferation.
c) The Ames Assay Test: The SOS response is utilized in the Ames Assay Test, a standard test whereby chemical toxicity is determined. According to the procedure, bacteria are exposed to a chemical solution in question, and if a genetic recombination accelerates via the spontaneous proliferation of viruses from these bacteria, then the chemical is determined to be a poison. The phenomenon is analogous to a poker player with a bad hand who must request an exchange of cards and a reshuffled deck to improve the possibilities for survival. In the Ames Assay Test, bacteria are concerned with their genetic “hand” in order to improve their abilities to metabolize poisons, create utilizations for poisons, and shield against poisons. Thus they engage in this well-known phenomena of “gene shuffling,” facilitated by virus proliferation.
Thus, I propose that the poliovirus is a symbiotic (and possibly a dormant) virus that behaves in a manner suggested by the phenomenon found in the Ames Assay Test, a test used to determine toxicity.
One could object to this analogy on the grounds that because the Ames Test utilizes prokaryote cells (bacteria-like cells) rather than eukaryote cells (nucleus-containing cells that comprise multicellular tissue) and because it is asserted that poliovirus invokes damage by infecting eukaryote cells, the explanation is invalid. However, the evolution of eukaryotes includes structures and functions inherited from symbiotic unions of prokaryotes. Eukaryotes continue to possess to this day prokaryote functionality such as found in the genetic independence of the organelles within the eukaryote cells, such as mitochondria (Lynn Margulis and Dorion Sagan, What Is Life? (1995), and, Lynn Margulis, Dorion Sagan, Slanted Truths: Essays on Gaia, Symbiosis, and Evolution (1997)). Thus, generalizations derived from the Ames Test can contribute well to a valid hypothesis for the presence of poliovirus in “polio”.
d) Dormant Virus: When a cell is critically threatened by toxic chemicals (or radiation) it can invoke survival mechanisms (the SOS Response) such as the suspension of metabolism, or the activation of dormant viruses, triggering their proliferation from the cell — such viruses are said to be “dormant” or “latent”. These words are not my preference because the way that they are popularly used implies that viruses are only externally generated and are found in the cell in a condition of temporary rest (dormancy). In cyclical phenomena, such as the life cycle of the virus, the “starting point” is a political-philosophical decision. The orthodox virus image (possibly a projection of the orthodox mind) is of an external, selfish, non-living parasite that tricks cells into infecting themselves with the virus and then to replicate said virus with cell machinery. Dormant viruses are publicized as external life forms that spend most of their time (as much as several decades) waiting inside cells, awaiting activation to perform parasitic activities.
Recently it has become known that a tremendous amount of human DNA is devoted to virus proliferation. The virologist, Eleni Papadopulos-Eleopulos, stated in Continuum, Autumn 1997:
…it’s accepted that endogenous retroviral DNA forms about 1% of human DNA… that’s about 3,000 times larger than what the experts claim is the size of the HIV genome. And what’s more, new retroviral genomes can arise by rearrangements and recombination of existing retroviral genomes.
Like the retroviruses, the poliovirus is an RNA virus and has a genome of similar weight and length. There is suspicion of dormant characteristics because enteroviruses have been found by several independent investigators, in post-polio (PMID: 8818905, UI: 96415998 (Lyon, France, Aug., 1996) and others).
e) Gene Sharing: Viruses represent shared capability, shared data, and data in transit. They are genetic couriers. Shared data decreases the burden on each cell to carry all capabilities. Capability, in the form of genetic information, can be stored in the environment as virus “gene packets”, and different capabilities can be stored in different cells, just as humans each have, to some degree, uncommon capabilities which are shared with the community as needed. In the microbiotic world, when a specific capability is needed, cells share genetic information from the dynamically changing universal library of free floating genetic material, such as exists in viruses, free organelles, symbiotic parasites, and free nucleic acid, in addition to straight sexual intercourse where nucleic acid is transferred directly form cell to cell. It could be said that cells can carry dormant genetic information in the form of nucleic acid and when that information is required, share it through the proliferation of viruses.
For example, in terms of disease, a symbiotic virus presence could be explained as a provider of cathartic capabilities or mechanisms, appropriate for various toxic or stressed environments. These cathartic mechanisms are manifested as disease symptoms, in the form of masses of sacrificed leucocytes, obviously found in boils, pimples, and pocks. Orthodoxy gives the label “transduction” to the processes of virus infection. Transduction is one of several modes of intercellular transport of genetic material, which allows for direct, laterally passed genetic data. Such data is routinely used to alter cell structure and metabolism modes dynamically, without engaging in the slower, more formal, sexual reproduction cycles.
The concept of the symbiotic virus is explained in Encyclopedia Britannica, Macropaedia (1990) p507:
Although viruses were originally discovered and characterized because of the diseases they cause, most viruses that infect bacteria, plants, and animals (including humans) do not cause disease. In fact, bacteriophages [bacteria viruses] may be helpful in that they rapidly transfer genetic information from one bacterium to another, and viruses of plants and animals may convey genetic information among similar species, aiding the survival of their hosts in hostile environments.
Britannica continues with a praise of industrial biotechnology, and abruptly converts the probable-present into a future-made-possible by dependent consumers:
This could in the future be true for humans as well. Recombinant DNA biotechnology may allow genetic defects to be repaired by injecting afflicted persons with harmless viruses that carry and integrate functional genes to supplant defective ones.
The implication is that humans are not part of nature, however, in the next sentence Britannica states that we humans may already utilize symbiotic viruses:
Such events may actually occur in nature in the transmission of “good” viruses from one person to another.
The nature-friendly view, that viruses are effective genetic symbionts, dilutes the market impact of genetic-based treatments alluded to by Britannica, and threatens biotech profits. Perhaps this explains certain aspects of the current worldwide “war” against virus-carrying mosquitoes?
The concept of a predatory poliovirus becomes less certain in the context of these little known virus “facts”:
|1.||Poliovirus “[I]nfectosomes have yet to be experimentally demonstrated…”, writes Roland R. Rueckert, under the subtitle, “Infection: A Rare Event” in Fields Virology.|
|2.||“Eukaryote cells have a wide arsenal of activities to control the half-lives of mRNAs, and these nucleases have made it difficult to isolate intact RNA viral genomes from cells.” (“Virus Evolution”, Ellen G. Strauss, et al, Fields Virology, Lippincott – Raven Publishers, Philadelphia (1996), v1p163)
In view of item 1, Rueckert, this appears to be another careful way of saying “never”.
|3.||The poliovirus does not always infect in accordance to its notoriety, “For every 200 or so virus particles that encounter a cell, only one will successfully enter and replicate, so research in this area is often confounded by the rarity of successful entry.” (http://cumicro2.cpmc.columbia.edu/PICO/Chapters/Cellular.html (defunct link))|
|4.||Only herpesvirus has been traced enroute to site of disease from site of infection. “Viruses during retrograde transport on their way up to the cell bodies have so far been localized ultrastructurally only in the case of herpes simplex and herpes virus suis.” (Martin E. Schwab and Hans Thoenen, Encyclopedia of Neuroscience, edited by George Adelman, pub, Birkhaüser Bros. Inc., Boston, 1987, Chapter 39, p102-3)|
|5.||A “poliovirus” has been electrophotographed in cell tissue. Due the lack of any photos of the virus as an infectosome, these photos should be interpreted as evidence of the cell’s SOS response rather than of poliovirus causation. Electrophotography has existed for several decades and has yet to photograph a poliovirus infectosome. An infectosome is a “membrane-associated particle… which transfers genomic viral RNA through the membrane.” (Field’s Virology, 1996, p635)|
|6.||“It seems likely that all viruses trace their origins to cellular genes and can be considered as pieces of rogue nucleic acids.” (Encyclopedia Britannica, Micropaedia, 1997, “Virus”)
This demonstrates the great potential for a symbiotic relation between viruses and “hosts”.
|7.||The point in history when known viruses began their evolution has been calculated by molecular biochemists who have interpolated backwards through time the speed and direction of virus evolution. They found that “most viruses we know today have probably evolved since the last ice age.” (“Virus Evolution”, Ellen G. Strauss, et al, Fields Virology, 1996, p164)|
|8.||Viruses are involved in a process called transduction, one of the three modes of genetic transfer between cells, a process that can accelerate genetic recombination when cells are critically threatened by poisons.|
|9.||Virus infection is used by clone technology to transfer genetic material into cells.|
|10.||“Genetic information moves between viruses and their hosts to the point where definitions and classifications begin to blur.” (Fields Virology, 1996, p6)|
|11.||In terms of genetic similarity, “[T]here was a remarkable continuum…” from virus to host. (Fields Virology, 1996, p6)|
|12.||“Carrel (1926) was able to produce tumors resembling Rous’ sarcoma and transmissible by cell-free filtrates with indol, arsenic, or tar in chicken embryo. Carrel’s observations have been confirmed by other workers. Fischer (1926), by treating cultures of normal cells with arsenic obtained on one occasion a filtrable virus capable of causing tumors.” (Ralph R. Scobey, M.D., “Poliomyelitis Caused by Exogenous Virus?”, Science, v71, 1954)|
Any of the items listed above can be used to direct work towards a refreshing view of viropathology. For instance, Alexis Carrel and Albert Fischer’s experiments, in 1925-1926, preceded the discovery of the cellular SOS Response by decades. Their work is important in its impact on the basic tenants of viropathology, the contemporary proofs of virus causation, and definitions of immunity. Carrel, who happens to be one of the most recognized of all the Nobel Laureates, has stated without equivocation that the Rous sarcoma tumour is not infective, is caused by an agent within the cells themselves, yet is transmissible by cell-free Berkfeld filtrate of tumour extract. He states that the agent could not be a virus because of his assumption that a virus is an external, disease-causing, infectious entity. In retrospect such statements reveal the first (unrecognized) discovery of the dormant retrovirus. Carrel also clearly demonstrates poisoncausation for cancer. These landmark experiments are very simple, very clear, and totally ignored by orthodoxy.
If one views Carrel and Fischer as a reinforcement of the symbiotic virus paradigm, then two strong alternative views can be defined regarding work that has been based on injections:
Virus Disease: In the case of classical induction of disease by injection of extremely high quantities of virus, the alternative view would be that the presence of such quantities of virus serve as an informational context, a context that indicates imminent toxic death to naïve tissue, with an expected tissue reaction (disease). Or in other words, disease induction (via injection) is no more than an over-reaction (like jumping out of a window when someone yells “fire”) in terms of inflammation and catharsis (disease manifestations).
Immunity: In the case of the classical demonstration of immunity whereby surviving subjects are found immune to attempts to induce disease by subsequent injections of virus, the alternative view is — you can’t fool them twice.
Thus, a) inducement of disease by the injection of high-quantities of virus, and b) acquired immunity in survivors of these injections, can both be viewed as parlour tricks, though claimed to be demonstrations of virus causation for disease.
The word “virus” is ancient Latin, meaning “slime” or “poison”. Mainstream science admits that most viruses are harmless, yet the word “virus” adds to a biased and highly promoted language of fear regarding nature. Definitions of viruses range from “pathogenic” to “not usually pathogenic” — the more popular the media source, the more frightening the definition. Less fearful definitions would change the relationship between the medical industry and its “patients”.
Paradoxically, early virus studies considered virus filtrates to be a poison, not a microbe, thus the name virus. Today, we know that viruses are information.
Now, nearly a half-century later, the validity of Dr. Biskind’s work appears even more certain. Again, according to Biskind:
It was even known by 1945 that DDT is stored in the body fat of mammals and appears in the milk. With this foreknowledge the series of catastrophic events that followed the most intensive campaign of mass poisoning in known human history, should not have surprised the experts. Yet, far from admitting a causal relationship so obvious that in any other field of biology it would be instantly accepted, virtually the entire apparatus of communication, lay and scientific alike, has been devoted to denying, concealing, suppressing, distorting and attempts to convert into its opposite, the overwhelming evidence. Libel, slander and economic boycott have not been overlooked in this campaign.
The unique correlations between CNS disease and CNS poisons present a variety of research opportunities not only in medical science, but political science, philosophy, media studies, psychology, and sociology.,,,,,
Author’s note, 2015:
This article describes a view of polio, faithful to the tenets of the original article of June 2000. Research has continued through the present, however. For more information, an evolution of facts and concepts, books and articles, see http://harvoa.org
The intent herein is to provide an impartial, scholarly analysis of CNS disease and chemical causation. Current research priorities are for proof of poliovirus causation and/or proof that invalidates chemical causation.
Corrections, uncredited sources and/or copyright infractions, if any, will be rectified upon notice. This site is not a monologue of truth. It is a catalyst for the reevaluation of “polio”. The reader is urged to confront officials to clarify issues mentioned herein.
This site is designed for critical, literary, and academic usage. A qualified and trustworthy medical professional must be consulted regarding medical issues, treatments, diagnoses, etc.
Once a local fetish, the cold-brew coffee trend kicked off in earnest in the summer of 2014 and expanded to become an international coffee movement. But questions remain about whether the brew process, which involves soaking coffee grounds in cold or room-temperature water for long periods, benefits consumers beyond the subjective issue of taste.
“As the market has picked up, marketers are beating consumers over the head on all sides with claims about the quality and sometimes unsubstantiated health benefits of RTD cold brew,” says Nick Brown, editor of Roast Magazine’s Daily Coffee News.
Those health claims include lower caffeine and acidity from a process that simply replaces the heat of a regular brew with time.
So How Does Cold-Brew Coffee Work?
The method creates coffee concentrate by soaking grounds in cold or room-temperature water for 12 to 24 hours. The filtered final product is diluted with two or three parts hot or cold water and milk or dairy substitute to one part coffee. The concentrate, or “essence,” can also be stored in the refrigerator for up to six weeks or frozen for a longer period.
Not to be confused with traditional iced coffee, in which hot-brewed coffee is poured over ice, cold-brew coffee is the product of a process that some claim can trace its early history to Dutch traders in the 1600s looking for a way of producing coffee that traveled easily. Others believe the practice began in Japan or Central and South America, according to DailyCoffeeNews.com.
Cold-Brew Coffee’s Health Claims
Coffee made from cold-brew concentrates has been credited with containing less caffeine and less acid than coffee made in the traditional hot-brewing method.
In tests using Starbucks’ regular coffee blend, researchers engaged by cold-brew system maker Toddy found that the cold-brew coffee had 40 milligrams of caffeine per 100 grams, while a store-brewed coffee registered at 61 milligrams of caffeine.
Large amounts of caffeine may lead to osteoporosis or fibrocystic disease, according to the National Institutes of Health. The NIH also warns against too much caffeine for pregnant women and children.
The Toddy study also found that regular coffee brewed cold had a pH of 6.31, as opposed to a 5.48 pH for the hot-brewed version. (On the pH scale lower numbers denote more acid.)
A diet lower in acid is credited with various benefits that would make cold-brewed coffee, if its low-acidity claims are true, appeal to health-conscious consumers. Some of those benefits may include supporting bone health, reducing muscle wasting and lessening the severity or incidence of hypertension and strokes, according to an article in the Journal of Environmental and Public Health called “The Alkaline Diet: Is There Evidence That an Alkaline pH Diet Benefits Health?”
In addition, the report noted, reversing the decrease in growth hormone caused by an acidic diet may benefit everything from cardiovascular health to memory and cognition.
But is that enough evidence to give up your coffeemaker?
“Nearly every hot- and cold-brewing method is different from the next, using different coffees, different ratios, different roasts and different recipes,” Brown notes. Plus, cold brew’s lower-acidity claims can generally be traced to the study commissioned by Toddy, Brown says, and the difference hasn’t been researched enough by the scientific community.
Fitness expert Jillian Michaels recently invested in Lucky Jack Organic Coffee Co., which makes a bottled organic cold-brew coffee infused with nitrogen.
The former coach on NBC’s “Biggest Loser” told Forbes.com contributor Robin D. Schatz that cholesterol concerns are one reason she decided to get into the business: “I’m a coffee person. But there are some issues with coffee. For example, coffee is the second-highest sprayed crop in the world. In addition, how you brew your coffee can have an impact your health. Studies have shown us with coffee that’s hot-brewed, some individuals can actually raise their LDL levels, which is also bad.”
A rise in cholesterol may not be limited to hot-brewed coffee, however. Cafestol and kahweol, chemical compounds known as diterpenes, are a natural part of the oils of the coffee, but they “raise the serum concentration of cholesterol and triglycerides in humans, and they also appear mildly to affect the integrity of liver cells,” according to a 1996 paper presented in the Journal of the Royal Society of Medicine.
Filtering coffee removes cafestol and kahweol. Yet since coffee is soluble and cold water doesn’t dissolve coffee as well as hot water — which contributes to cold brew’s less acidic taste — it’s possible that certain compounds don’t leach out of the coffee grounds when brewed with cold water.
More study is needed to determine if cold-brewing removes or inhibits the diterpenes, so you may want to also filter your cold-brew essence before consuming it if you have cholesterol concerns.
Heart Disease, Cancer and More
Coffee generally has been shown to be rich in antioxidants that may help prevent aging-related ailments like heart disease, Alzheimer’s and cancer. While you might have other reasons to love your cold brew, there is no evidence to suggest that the cold-brew process negates or enhances those antioxidants.
The process of oxidation, according to UCLA’s Science and Food, happens at a higher rate when coffee is brewed hot. Oxidized coffee oils can cause coffee to taste bitter and sour, but whether the high temperature also reduces coffee’s antioxidant benefits has not yet been fully researched.
Read the Label on Ready-to-Drink Packaged Cold Brews
Ready-to-drink (RTD) products might be shelf-stabilized in ways home-brewed drinks aren’t, Brown says. He recommends that consumers scour product labels for ingredients they would normally avoid in any other drink or processed food.
“There’s a tremendous and quite confusing variety of RTD products out there, with more coming every day,” Brown says. “The cold RTD beverage segment as a whole has been growing strong, and I’m sure cold brew, being produced largely by coffee roasters, has gotten a bump from the kind of American ‘craft food’ movement.”
Market observers credit the rise of RTD products to companies like Portland-based Stumptown, which began selling its packaged cold-brew coffee in 2011.
The trend has since proved its staying power, with big-brand coffee retailers Starbucks and Peet’s along with grocer Trader Joe’s getting in on the action. Peet’s, which replaced its traditional iced-coffee line with cold brew in June 2015, acquired Stumptown in October that year.
Other Possible Benefits
The cold-brew method would also seem to save a few pennies on electricity — and the physical process of producing a large jug of cold brew may well burn a few calories. Reuse your coffee grounds in your garden or for a body scrub to make cold-brew an all-around more eco-friendly choice.
What Do YOU Think?
Are you a fan of cold-brew coffee? Why or why not? What are your thoughts on the health claims about this new trend? Tell us in the comments!